Soluble amyloid-β oligomers as synaptotoxins leading to cognitive impairment in Alzheimer’s disease

Frontiers in Cellular Neuroscience

Volumn 9, Issue MAY, 2015, Pages

  Ferreira, Sergio T ^a   Lourenco, Mychael V ^a   Oliveira, Mauricio M ^a   De Felice, Fernanda G ^a  

^a FEDERAL UNIVERSITY OF RIO DE JANEIRO   (Brazil)

Author keywords

Alzheimer s disease; Amyloid oligomers; Memory loss; Neuronal dysfunction; Synapse failure

Indexed keywords

ADUCANUMAB; AMYLOID BETA PROTEIN; FRIZZLED PROTEIN; GLUTAMATE RECEPTOR; NEUROLIGIN 1; OLIGOMER; PRION PROTEIN; SIGMA 2 OPIATE RECEPTOR; TAU PROTEIN;

ALZHEIMER DISEASE; BINDING AFFINITY; COGNITIVE DEFECT; GLIA CELL; HUMAN; MEMORY DISORDER; MOLECULAR WEIGHT; NERVE DEGENERATION; NONHUMAN; PATHOGENESIS; PROTEIN EXPRESSION; PROTEIN INTERACTION; PROTEIN PHOSPHORYLATION; REVIEW; SIGNAL TRANSDUCTION; SYNAPSE;

EID: 84930504118     PISSN: 16625102     EISSN: None     Source Type: Journal    
DOI: 10.3389/fncel.2015.00191     Document Type: Review

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