Ubiquitin Hydrolase Uch-L1 Rescues β-Amyloid-Induced Decreases in Synaptic Function and Contextual Memory

Cell

Volumn 126, Issue 4, 2006, Pages 775-788

  Gong, Bing ^a   Cao, Zixuan ^a   Zheng, Ping ^b   Vitolo, Ottavio V ^a   Liu, Shumin ^a   Staniszewski, Agnieszka ^a   Moolman, Donna ^a   Zhang, Hong ^a   Shelanski, Michael ^a   Arancio, Ottavio ^a  

^a Och Spine at New York Presbyterian Hospitals   (United States)

^b NYU HOSPITAL FOR JOINT DISEASES   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

AMYLOID BETA PROTEIN; CYCLIC AMP DEPENDENT PROTEIN KINASE; CYCLIC AMP RESPONSIVE ELEMENT BINDING PROTEIN; HYBRID PROTEIN; OLIGOMER; PROTEIN DERIVATIVE; TRANSACTIVATOR PROTEIN; UBIQUITIN C TERMINAL HYDROLASE L1; UNCLASSIFIED DRUG;

ALZHEIMER DISEASE; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN SLICE; CARBOXY TERMINAL SEQUENCE; COGNITION; CONTROLLED STUDY; ENZYME ACTIVITY; HIPPOCAMPUS; LEARNING; MEMORY; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN DOMAIN; PROTEIN FUNCTION; PROTEIN PHOSPHORYLATION; SYNAPSE;

EID: 33747199933     PISSN: 00928674     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.cell.2006.06.046     Document Type: Article

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