Epigenetic suppression of neuroligin 1 underlies amyloid-induced memory deficiency

Nature Neuroscience

Volumn 17, Issue 2, 2014, Pages 223-231

  Bie, Bihua ^a   Wu, Jiang ^a   Yang, Hui ^a   Xu, Jijun J ^a   Brown, David L ^a   Naguib, Mohamed ^a  

^a LERNER RESEARCH INSTITUTE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

AMYLOID; CYTOSINE; HISTONE DEACETYLASE 2; HISTONE H3; METHYL CPG BINDING PROTEIN 2; NEUROLIGIN 1;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CONTROLLED STUDY; HIPPOCAMPAL CA1 REGION; HISTONE ACETYLATION; MALE; MEMORY; MEMORY DISORDER; METHYLATION; MICROGLIA; MOUSE; NERVOUS SYSTEM INFLAMMATION; NONHUMAN; PATHOGENESIS; PRIORITY JOURNAL; PROTEIN PROTEIN INTERACTION; RAT; SYNAPTIC EFFICACY;

AMYLOID; AMYLOID BETA-PROTEIN PRECURSOR; ANIMALS; CELL ADHESION MOLECULES, NEURONAL; DISEASE MODELS, ANIMAL; DNA METHYLATION; EXCITATORY AMINO ACID ANTAGONISTS; HIPPOCAMPUS; HISTONE DEACETYLASE 2; HUMANS; LIPOPOLYSACCHARIDES; MALE; MAZE LEARNING; MEMBRANE POTENTIALS; MEMORY DISORDERS; METHYL-CPG-BINDING PROTEIN 2; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; PRESENILIN-1; RATS; RATS, SPRAGUE-DAWLEY; TIME FACTORS;

EID: 84893751693     PISSN: 10976256     EISSN: 15461726     Source Type: Journal    
DOI: 10.1038/nn.3618     Document Type: Article

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