The two faces of protein misfolding: Gain- and loss-of-function in neurodegenerative diseases

EMBO Journal

Volumn 27, Issue 2, 2008, Pages 336-349

  Winklhofer, Konstanze F ^a   Tatzelt, Jörg ^a   Haass, Christian ^a  

^a UNIVERSITY OF MUNICH   (Germany)

Author keywords

Alzheimer; FTLD; Misfolding; Parkinson; Prion

Indexed keywords

ALPHA SYNUCLEIN; AMYLOID BETA PROTEIN; OLIGOMER; PARKIN; TAU PROTEIN; UBIQUITIN PROTEIN LIGASE E3;

ALZHEIMER DISEASE; CHROMOSOME 17; DEGENERATIVE DISEASE; FRONTOTEMPORAL DEMENTIA; GENE IDENTIFICATION; HUMAN; MOLECULAR BIOLOGY; NERVE CELL DEGENERATION; NERVE DEGENERATION; NEUROPATHOLOGY; NEUROPROTECTION; NONHUMAN; OXIDATIVE STRESS; PARKINSON DISEASE; PRION DISEASE; PRIORITY JOURNAL; PROTEIN ASSEMBLY; PROTEIN BINDING; PROTEIN FOLDING; REVIEW;

ALZHEIMER DISEASE; AMYLOID BETA-PROTEIN; HUMANS; MODELS, BIOLOGICAL; NEURODEGENERATIVE DISEASES; PARKINSON DISEASE; PROTEIN FOLDING; PROTEINS;

ANIMALIA;

EID: 38549169530     PISSN: 02614189     EISSN: 14602075     Source Type: Journal    
DOI: 10.1038/sj.emboj.7601930     Document Type: Review

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