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Volumn 98, Issue 26, 2001, Pages 14955-14960
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Wild-type PrP and a mutant associated with prion disease are subject to retrograde transport and proteasome degradation
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Author keywords
[No Author keywords available]
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Indexed keywords
EPOXOMICIN;
PLASMID DNA;
PRION PROTEIN;
PROTEASOME;
ANIMAL CELL;
ARTICLE;
CELL STRAIN COS1;
CENTROSOME;
CONTROLLED STUDY;
CYTOPLASM;
ENDOPLASMIC RETICULUM;
ENZYME ACTIVITY;
ENZYME DEGRADATION;
HUMAN;
HUMAN CELL;
IMMUNOFLUORESCENCE;
NEUROBLASTOMA CELL;
NONHUMAN;
PATHOGENESIS;
PRION DISEASE;
PRIORITY JOURNAL;
PROTEIN AGGREGATION;
PROTEIN CONFORMATION;
PROTEIN DEGRADATION;
PROTEIN PROCESSING;
RETROGRADE NERVE FIBER TRANSPORT;
BASE SEQUENCE;
CELL LINE;
CYSTEINE ENDOPEPTIDASES;
CYSTEINE PROTEINASE INHIBITORS;
DNA PRIMERS;
FLUORESCENT ANTIBODY TECHNIQUE;
HYDROLYSIS;
MULTIENZYME COMPLEXES;
MUTAGENESIS, SITE-DIRECTED;
PRION DISEASES;
PROTEASOME ENDOPEPTIDASE COMPLEX;
PROTEIN TRANSPORT;
PRPSC PROTEINS;
ANIMALIA;
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EID: 0035910069
PISSN: 00278424
EISSN: None
Source Type: Journal
DOI: 10.1073/pnas.011578098 Document Type: Article |
Times cited : (277)
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References (35)
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