Physico-Pathologic Mechanisms Involved in Neurodegeneration: Misfolded Protein-Plasma Membrane Interactions

Neuron

Volumn 95, Issue 1, 2017, Pages 33-50

  Shrivastava, Amulya Nidhi ^a,b   Aperia, Anita ^c   Melki, Ronald ^b   Triller, Antoine ^a  

^a PSL RESEARCH UNIVERSITY   (France)

^b CNRS   (France)

^c KAROLINSKA INSTITUTE   (Sweden)

Author keywords

Alzheimer; membrane interaction; Parkinson; pathogenic proteins; prion like propagation; protein diffusion; protein aggregation

Indexed keywords

GLUTAMATE RECEPTOR; LIPID; MEMBRANE PROTEIN; ORGANIC CATION TRANSPORTER; PROTEIN; SCAFFOLD PROTEIN; ALPHA SYNUCLEIN; AMYLOID BETA PROTEIN; COPPER ZINC SUPEROXIDE DISMUTASE; HUNTINGTIN; PRION; TAU PROTEIN;

ALZHEIMER DISEASE; AMYOTROPHIC LATERAL SCLEROSIS; CELL MEMBRANE; CELL MIGRATION; CHANNEL GATING; EXTRACELLULAR MATRIX; HUMAN; HUNTINGTON CHOREA; NERVE DEGENERATION; NONHUMAN; PARKINSON DISEASE; PATHOGENESIS; PRIORITY JOURNAL; PROTEIN AGGREGATION; PROTEIN ASSEMBLY; PROTEIN INTERACTION; PROTEIN LIPID INTERACTION; PROTEIN LOCALIZATION; PROTEIN MISFOLDING; PROTEIN STRUCTURE; REVIEW; ANIMAL; DEGENERATIVE DISEASE; EXTRACELLULAR SPACE; METABOLISM; PRION; PROTEIN FOLDING; PROTEIN TRANSPORT; PROTEINOSIS;

ALPHA-SYNUCLEIN; ALZHEIMER DISEASE; AMYLOID BETA-PEPTIDES; AMYOTROPHIC LATERAL SCLEROSIS; ANIMALS; CELL MEMBRANE; EXTRACELLULAR SPACE; HUMANS; HUNTINGTIN PROTEIN; HUNTINGTON DISEASE; NEURODEGENERATIVE DISEASES; PARKINSON DISEASE; PRIONS; PROTEIN AGGREGATION, PATHOLOGICAL; PROTEIN FOLDING; PROTEIN TRANSPORT; SUPEROXIDE DISMUTASE-1; TAU PROTEINS;

EID: 85027275966     PISSN: 08966273     EISSN: 10974199     Source Type: Journal    
DOI: 10.1016/j.neuron.2017.05.026     Document Type: Review

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