Mitochondrial pathways, permeability transition pore, and redox signaling in cardioprotection: Therapeutic implications

Antioxidants and Redox Signaling

Volumn 18, Issue 5, 2013, Pages 556-599

  Penna, Claudia ^a,b   Perrelli, Maria Giulia ^a,b   Pagliaro, Pasquale ^a,b  

^a UNIVERSITY OF TURIN   (Italy)

^b National Institute of Cardiovascular Research INRC   (Italy)

Author keywords

[No Author keywords available]

Indexed keywords

ADENOSINE TRIPHOSPHATE; ANTIOXIDANT; CYCLOSPORIN A; GLYCOGEN SYNTHASE KINASE 3BETA; HYDROXYMETHYLGLUTARYL COENZYME A REDUCTASE KINASE; MITOCHONDRIAL PERMEABILITY TRANSITION PORE; NITROGEN DERIVATIVE; PHOSPHOTRANSFERASE; POTASSIUM CHANNEL; PROTEIN BCL 2; PROTEIN KINASE C; PROTEIN KINASE PIM 1; REACTIVE OXYGEN METABOLITE; STAT3 PROTEIN;

ACUTE HEART INFARCTION; AGING; ANGIOPLASTY; AUTOPHAGY; CELL SURVIVAL; CELL VIABILITY; CELLULAR STRESS RESPONSE; COMORBIDITY; ENZYME ACTIVATION; HEART INFARCTION PREVENTION; HEART MUSCLE CELL; HEART MUSCLE INJURY; HEART MUSCLE ISCHEMIA; HEART MUSCLE REPERFUSION; HEART PROTECTION; HUMAN; HYPERTENSION; HYPERTROPHY; ISCHEMIC POSTCONDITIONING; MITOCHONDRION; MITOPHAGY; NONHUMAN; OXIDATION REDUCTION REACTION; OXIDATIVE PHOSPHORYLATION UNCOUPLING; OXIDATIVE STRESS; PRIORITY JOURNAL; REPERFUSION INJURY; REVIEW; RNA INTERFERENCE; SIGNAL TRANSDUCTION;

ANIMALS; AUTOPHAGY; CARDIOTONIC AGENTS; HUMANS; MITOCHONDRIA, HEART; MITOCHONDRIAL DEGRADATION; MYOCARDIAL REPERFUSION INJURY; OXIDATION-REDUCTION; REACTIVE OXYGEN SPECIES; SIGNAL TRANSDUCTION;

EID: 84872182382     PISSN: 15230864     EISSN: 15577716     Source Type: Journal    
DOI: 10.1089/ars.2011.4459     Document Type: Review

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