-
1
-
-
0037383322
-
GSK-3: tricks of the trade for a multi-tasking kinase
-
Doble B.W., and Woodgett J.R. GSK-3: tricks of the trade for a multi-tasking kinase. J. Cell. Sci. 116 (2003) 1175-1186
-
(2003)
J. Cell. Sci.
, vol.116
, pp. 1175-1186
-
-
Doble, B.W.1
Woodgett, J.R.2
-
2
-
-
33748299477
-
The paradoxical pro- and anti-apoptotic actions of GSK3 in the intrinsic and extrinsic apoptosis signaling pathways
-
Beurel E., and Jope R.S. The paradoxical pro- and anti-apoptotic actions of GSK3 in the intrinsic and extrinsic apoptosis signaling pathways. Prog. Neurobiol. 79 (2006) 173-189
-
(2006)
Prog. Neurobiol.
, vol.79
, pp. 173-189
-
-
Beurel, E.1
Jope, R.S.2
-
3
-
-
0037040856
-
Phosphorylation of glycogen synthase kinase-3beta during preconditioning through a phosphatidylinositol-3-kinase-dependent pathway is cardioprotective
-
Tong H., Imahashi K., Steenbergen C., and Murphy E. Phosphorylation of glycogen synthase kinase-3beta during preconditioning through a phosphatidylinositol-3-kinase-dependent pathway is cardioprotective. Circ. Res. 90 (2002) 377-379
-
(2002)
Circ. Res.
, vol.90
, pp. 377-379
-
-
Tong, H.1
Imahashi, K.2
Steenbergen, C.3
Murphy, E.4
-
4
-
-
33746837190
-
Erythropoietin affords additional cardioprotection to preconditioned hearts by enhanced phosphorylation of glycogen synthase kinase-3 beta
-
Nishihara M., Miura T., Miki T., Sakamoto J., Tanno M., Kobayashi H., et al. Erythropoietin affords additional cardioprotection to preconditioned hearts by enhanced phosphorylation of glycogen synthase kinase-3 beta. Am. J. Physiol. 29 (2006) H748-H755
-
(2006)
Am. J. Physiol.
, vol.29
-
-
Nishihara, M.1
Miura, T.2
Miki, T.3
Sakamoto, J.4
Tanno, M.5
Kobayashi, H.6
-
5
-
-
1842841258
-
Opioid-induced cardioprotection occurs via glycogen synthase kinase beta inhibition during reperfusion in intact rat hearts
-
Gross E.R., Hsu A.K., and Gross G.J. Opioid-induced cardioprotection occurs via glycogen synthase kinase beta inhibition during reperfusion in intact rat hearts. Circ. Res. 94 (2004) 960-966
-
(2004)
Circ. Res.
, vol.94
, pp. 960-966
-
-
Gross, E.R.1
Hsu, A.K.2
Gross, G.J.3
-
6
-
-
33646116079
-
Bradykinin prevents reperfusion injury by targeting mitochondrial permeability transition pore through glycogen synthase kinase 3beta
-
Park S.S., Zhao H., Mueller R.A., and Xu Z. Bradykinin prevents reperfusion injury by targeting mitochondrial permeability transition pore through glycogen synthase kinase 3beta. J. Mol. Cell. Cardiol. 40 (2006) 708-716
-
(2006)
J. Mol. Cell. Cardiol.
, vol.40
, pp. 708-716
-
-
Park, S.S.1
Zhao, H.2
Mueller, R.A.3
Xu, Z.4
-
7
-
-
33745273909
-
6-(3-Iodobenzyl)-adenosine-5′-N-methylcarboxamide confers cardioprotection at reperfusion by inhibiting mitochondrial permeability transition pore opening via glycogen synthase kinase 3 beta
-
6-(3-Iodobenzyl)-adenosine-5′-N-methylcarboxamide confers cardioprotection at reperfusion by inhibiting mitochondrial permeability transition pore opening via glycogen synthase kinase 3 beta. J. Pharmacol. Exp. Ther. 318 (2006) 124-131
-
(2006)
J. Pharmacol. Exp. Ther.
, vol.318
, pp. 124-131
-
-
Park, S.S.1
Zhao, H.2
Jang, Y.3
Mueller, R.A.4
Xu, Z.5
-
8
-
-
33745234137
-
NECA at reperfusion limits infarction and inhibits formation of the mitochondrial permeability transition pore by activating p70S6 kinase
-
Forster K., Paul I., Solenkova N., Staudt A., Cohen M.V., Downey J.M., et al. NECA at reperfusion limits infarction and inhibits formation of the mitochondrial permeability transition pore by activating p70S6 kinase. Basic Res. Cardiol. 101 (2006) 319-326
-
(2006)
Basic Res. Cardiol.
, vol.101
, pp. 319-326
-
-
Forster, K.1
Paul, I.2
Solenkova, N.3
Staudt, A.4
Cohen, M.V.5
Downey, J.M.6
-
9
-
-
85047692700
-
Glycogen synthase kinase-3beta mediates convergence of protection signaling to inhibit the mitochondrial permeability transition pore
-
Juhaszova M., Zorov D.B., Kim S.H., Pepe S., Fu Q., Fishbein K.W., et al. Glycogen synthase kinase-3beta mediates convergence of protection signaling to inhibit the mitochondrial permeability transition pore. J. Clin. Invest. 113 (2004) 1535-1549
-
(2004)
J. Clin. Invest.
, vol.113
, pp. 1535-1549
-
-
Juhaszova, M.1
Zorov, D.B.2
Kim, S.H.3
Pepe, S.4
Fu, Q.5
Fishbein, K.W.6
-
10
-
-
1142273368
-
Mitochondrial permeability transition pore opening during myocardial reperfusion-A target for cardioprotection
-
Halestrap A.P., Clarke S.J., and Javadov S.A. Mitochondrial permeability transition pore opening during myocardial reperfusion-A target for cardioprotection. Cardiovasc. Res. 61 (2004) 372-385
-
(2004)
Cardiovasc. Res.
, vol.61
, pp. 372-385
-
-
Halestrap, A.P.1
Clarke, S.J.2
Javadov, S.A.3
-
11
-
-
16344374718
-
Mitochondrial function and myocardial aging. A critical analysis of the role of permeability transition
-
Di Lisa F., and Bernardi P. Mitochondrial function and myocardial aging. A critical analysis of the role of permeability transition. Cardiovasc. Res. 66 (2005) 222-232
-
(2005)
Cardiovasc. Res.
, vol.66
, pp. 222-232
-
-
Di Lisa, F.1
Bernardi, P.2
-
12
-
-
0036023641
-
Inhibiting mitochondrial permeability transition pore opening: a new paradigm for myocardial preconditioning?
-
Hausenloy D.J., Maddock H.L., Baxter G.F., and Yellon D.M. Inhibiting mitochondrial permeability transition pore opening: a new paradigm for myocardial preconditioning?. Cardiovasc. Res. 55 (2002) 534-543
-
(2002)
Cardiovasc. Res.
, vol.55
, pp. 534-543
-
-
Hausenloy, D.J.1
Maddock, H.L.2
Baxter, G.F.3
Yellon, D.M.4
-
13
-
-
0038172269
-
Ischaemic preconditioning inhibits opening of mitochondrial permeability transition pores in the reperfused rat heart
-
Javadov S.A., Clarke S., Das M., Griffiths E.J., Lim K.H., and Halestrap A.P. Ischaemic preconditioning inhibits opening of mitochondrial permeability transition pores in the reperfused rat heart. J. Physiol. 549 (2003) 513-524
-
(2003)
J. Physiol.
, vol.549
, pp. 513-524
-
-
Javadov, S.A.1
Clarke, S.2
Das, M.3
Griffiths, E.J.4
Lim, K.H.5
Halestrap, A.P.6
-
14
-
-
33645118786
-
Alteration in erythropoietin-induced cardioprotective signaling by postinfarct ventricular remodeling
-
Miki T., Miura T., Yano T., Takahashi A., Sakamoto J., Tanno M., et al. Alteration in erythropoietin-induced cardioprotective signaling by postinfarct ventricular remodeling. J. Pharmacol. Exp. Ther. 317 (2006) 68-75
-
(2006)
J. Pharmacol. Exp. Ther.
, vol.317
, pp. 68-75
-
-
Miki, T.1
Miura, T.2
Yano, T.3
Takahashi, A.4
Sakamoto, J.5
Tanno, M.6
-
15
-
-
33846694888
-
Impairment of cardioprotective PI3K-Akt signaling by post-infarct ventricular remodeling is compensated by an ERK-mediated pathway
-
Miki T., Miura T., Tanno M., Nishihara M., Naitoh K., Sato T., et al. Impairment of cardioprotective PI3K-Akt signaling by post-infarct ventricular remodeling is compensated by an ERK-mediated pathway. Basic Res. Cardiol. 102 (2007) 163-170
-
(2007)
Basic Res. Cardiol.
, vol.102
, pp. 163-170
-
-
Miki, T.1
Miura, T.2
Tanno, M.3
Nishihara, M.4
Naitoh, K.5
Sato, T.6
-
16
-
-
0035903235
-
Bid is cleaved by calpain to an active fragment in vitro and during myocardial ischemia/reperfusion
-
Chen M., He H., Zhan S., Krajewski S., Reed J.C., and Gottlieb R.A. Bid is cleaved by calpain to an active fragment in vitro and during myocardial ischemia/reperfusion. J. Biol. Chem. 276 (2001) 30724-30728
-
(2001)
J. Biol. Chem.
, vol.276
, pp. 30724-30728
-
-
Chen, M.1
He, H.2
Zhan, S.3
Krajewski, S.4
Reed, J.C.5
Gottlieb, R.A.6
-
19
-
-
34548437020
-
δ-Opioid receptor activation before ischemia reduces gap junction permeability in ischemic myocardium by PKC-ε-mediated phosphorylation of connexin-43
-
Miura T., Yano T., Naitoh K., Nishihara M., Miki T., Tanno M., et al. δ-Opioid receptor activation before ischemia reduces gap junction permeability in ischemic myocardium by PKC-ε-mediated phosphorylation of connexin-43. Am. J. Physiol.: Heart Circ. Physiol. 293 (2007) 1425-1431
-
(2007)
Am. J. Physiol.: Heart Circ. Physiol.
, vol.293
, pp. 1425-1431
-
-
Miura, T.1
Yano, T.2
Naitoh, K.3
Nishihara, M.4
Miki, T.5
Tanno, M.6
-
20
-
-
0442290275
-
Glycogen synthase kinase-3 beta is highly activated in nuclei and mitochondria
-
Bijur G.N., and Jope R.S. Glycogen synthase kinase-3 beta is highly activated in nuclei and mitochondria. NeuroReport 14 (2003) 2415-2419
-
(2003)
NeuroReport
, vol.14
, pp. 2415-2419
-
-
Bijur, G.N.1
Jope, R.S.2
-
21
-
-
0442274617
-
Glycogen synthase kinase-3beta (GSK3beta) binds to and promotes the actions of p53
-
Watcharasit P., Bijur G.N., Song L., Zhu J., Chen X., and Jope R.S. Glycogen synthase kinase-3beta (GSK3beta) binds to and promotes the actions of p53. J. Biol. Chem. 278 (2003) 48872-48879
-
(2003)
J. Biol. Chem.
, vol.278
, pp. 48872-48879
-
-
Watcharasit, P.1
Bijur, G.N.2
Song, L.3
Zhu, J.4
Chen, X.5
Jope, R.S.6
-
22
-
-
7744227096
-
Glycogen synthase kinase-3beta phosphorylates Bax and promotes its mitochondrial localization during neuronal apoptosis
-
Linseman D.A., Butts B.D., Precht T.A., Phelps R.A., Le S.S., Laessig T.A., et al. Glycogen synthase kinase-3beta phosphorylates Bax and promotes its mitochondrial localization during neuronal apoptosis. J. Neurosci. 24 (2004) 9993-10002
-
(2004)
J. Neurosci.
, vol.24
, pp. 9993-10002
-
-
Linseman, D.A.1
Butts, B.D.2
Precht, T.A.3
Phelps, R.A.4
Le, S.S.5
Laessig, T.A.6
-
23
-
-
33644855216
-
Glycogen synthase kinase-3 regulates mitochondrial outer membrane permeabilization and apoptosis by destabilization of MCL-1
-
Maurer U., Charvet C., Wagman A.S., Dejardin E., and Green D.R. Glycogen synthase kinase-3 regulates mitochondrial outer membrane permeabilization and apoptosis by destabilization of MCL-1. Mol. Cell 21 (2006) 749-760
-
(2006)
Mol. Cell
, vol.21
, pp. 749-760
-
-
Maurer, U.1
Charvet, C.2
Wagman, A.S.3
Dejardin, E.4
Green, D.R.5
-
24
-
-
33646034941
-
Acute PKCδ inhibition limits ischaemia-reperfusion injury in the aged rat heart: role of GSK-3β
-
Kostyak J.C., Hunter J.C., and Korzick D.H. Acute PKCδ inhibition limits ischaemia-reperfusion injury in the aged rat heart: role of GSK-3β. Cardiovasc. Res. 70 (2006) 325-334
-
(2006)
Cardiovasc. Res.
, vol.70
, pp. 325-334
-
-
Kostyak, J.C.1
Hunter, J.C.2
Korzick, D.H.3
-
25
-
-
33746207800
-
Translocation of connexin 43 to the inner mitochondrial membrane of cardiomyocytes through the heat shock protein 90-dependent TOM pathway and its importance for cardioprotection
-
Rodriguez-Sinovas A., Boengler K., Cabestrero A., Gres P., Morente M., Ruiz-Meana M., et al. Translocation of connexin 43 to the inner mitochondrial membrane of cardiomyocytes through the heat shock protein 90-dependent TOM pathway and its importance for cardioprotection. Circ. Res. 99 (2006) 93-101
-
(2006)
Circ. Res.
, vol.99
, pp. 93-101
-
-
Rodriguez-Sinovas, A.1
Boengler, K.2
Cabestrero, A.3
Gres, P.4
Morente, M.5
Ruiz-Meana, M.6
-
26
-
-
0037144409
-
Sanglifehrin A acts as a potent inhibitor of the mitochondrial permeability transition and reperfusion injury of the heart by binding to cyclophilin-D at a different site from cyclosporine A
-
Clarke S.J., McStay G.P., and Halestrap A.P. Sanglifehrin A acts as a potent inhibitor of the mitochondrial permeability transition and reperfusion injury of the heart by binding to cyclophilin-D at a different site from cyclosporine A. J. Biol. Chem. 277 (2002) 34793-34799
-
(2002)
J. Biol. Chem.
, vol.277
, pp. 34793-34799
-
-
Clarke, S.J.1
McStay, G.P.2
Halestrap, A.P.3
-
27
-
-
6344287496
-
Cyclophilin-D promotes the mitochondrial permeability transition but has opposite effects on apoptosis and necrosis
-
Li Y., Johnson N., Capano M., Edwards M., and Crompton M. Cyclophilin-D promotes the mitochondrial permeability transition but has opposite effects on apoptosis and necrosis. Biochem. J. 383 (2004) 101-109
-
(2004)
Biochem. J.
, vol.383
, pp. 101-109
-
-
Li, Y.1
Johnson, N.2
Capano, M.3
Edwards, M.4
Crompton, M.5
-
28
-
-
0032860116
-
Cyclosporin A and its nonimmunosuppressive analogue N-Me-Val-4-cyclosporin A mitigate glucose/oxygen deprivation-induced damage to rat cultured hippocampal neurons
-
Khaspekov L., Friberg H., Halestrap A., Viktorov I., and Wieloch T. Cyclosporin A and its nonimmunosuppressive analogue N-Me-Val-4-cyclosporin A mitigate glucose/oxygen deprivation-induced damage to rat cultured hippocampal neurons. Eur. J. Neurosci. 11 (1999) 3194-3198
-
(1999)
Eur. J. Neurosci.
, vol.11
, pp. 3194-3198
-
-
Khaspekov, L.1
Friberg, H.2
Halestrap, A.3
Viktorov, I.4
Wieloch, T.5
-
29
-
-
15844407874
-
Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death
-
Nakagawa T., Shimizu S., Watanabe T., Yamaguchi O., Otsu K., Yamagata H., et al. Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death. Nature 434 (2005) 652-658
-
(2005)
Nature
, vol.434
, pp. 652-658
-
-
Nakagawa, T.1
Shimizu, S.2
Watanabe, T.3
Yamaguchi, O.4
Otsu, K.5
Yamagata, H.6
-
30
-
-
15844375853
-
Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death
-
Baines C.P., Kaiser R.A., Purcell N.H., Blair N.S., Osinska H., Hambleton M.A., et al. Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death. Nature 434 (2005) 658-662
-
(2005)
Nature
, vol.434
, pp. 658-662
-
-
Baines, C.P.1
Kaiser, R.A.2
Purcell, N.H.3
Blair, N.S.4
Osinska, H.5
Hambleton, M.A.6
-
31
-
-
24744460273
-
Cyclophilin D is a component of mitochondrial permeability transition and mediates neuronal cell death after focal cerebral ischemia
-
Schinzel A.C., Takeuchi O., Huang Z., Fisher J.K., Zhou Z., Rubens J., et al. Cyclophilin D is a component of mitochondrial permeability transition and mediates neuronal cell death after focal cerebral ischemia. Proc. Natl. Acad. Sci. USA 102 (2005) 12005-12010
-
(2005)
Proc. Natl. Acad. Sci. USA
, vol.102
, pp. 12005-12010
-
-
Schinzel, A.C.1
Takeuchi, O.2
Huang, Z.3
Fisher, J.K.4
Zhou, Z.5
Rubens, J.6
-
32
-
-
34247895697
-
Voltage-dependent anion channels are dispensable for mitochondrial-dependent cell death
-
Baines C.P., Kaiser R.A., Sheiko T., Craigen W.J., and Molkentin J.D. Voltage-dependent anion channels are dispensable for mitochondrial-dependent cell death. Nat. Cell Biol. 9 (2007) 550-555
-
(2007)
Nat. Cell Biol.
, vol.9
, pp. 550-555
-
-
Baines, C.P.1
Kaiser, R.A.2
Sheiko, T.3
Craigen, W.J.4
Molkentin, J.D.5
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