Transient Mitochondrial Permeability Transition Pore Opening Mediates Preconditioning-Induced Protection

Circulation

Volumn 109, Issue 14, 2004, Pages 1714-1717

  Hausenloy, Derek ^a   Wynne, Abigail ^a   Duchen, Michael ^a   Yellon, Derek ^a  

^a UNIVERSITY COLLEGE LONDON   (United Kingdom)

Author keywords

Free radicals; Ischemia; Myocardial infarction; Reperfusion

Indexed keywords

2,4 DINITROPHENOL; CALCEIN; CYCLOSPORIN A; DIAZOXIDE; REACTIVE OXYGEN METABOLITE; SANGLIFEHRIN A; TIOPRONIN;

ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; HEART INFARCTION PREVENTION; HEART MUSCLE CELL; HEART MUSCLE ISCHEMIA; HEART MUSCLE REPERFUSION; HEART PROTECTION; ISOLATED HEART; MALE; MITOCHONDRION; NONHUMAN; POROSITY; PRIORITY JOURNAL; RAT;

2,4-DINITROPHENOL; ADENOSINE; ADRENERGIC ALPHA-AGONISTS; ANIMALS; CARDIOTONIC AGENTS; CELLS, CULTURED; CYCLOSPORINE; DIAZOXIDE; ELECTRIC CONDUCTIVITY; ION CHANNEL GATING; ION CHANNELS; ION TRANSPORT; ISCHEMIC PRECONDITIONING, MYOCARDIAL; LACTONES; MACROMOLECULAR SUBSTANCES; MALE; MITOCHONDRIA, HEART; MITOCHONDRIAL MEMBRANE TRANSPORT PROTEINS; MYOCARDIAL ISCHEMIA; MYOCARDIAL REPERFUSION INJURY; MYOCYTES, CARDIAC; OXIDATIVE PHOSPHORYLATION; RATS; RATS, SPRAGUE-DAWLEY; REACTIVE OXYGEN SPECIES; RECEPTORS, ADRENERGIC, ALPHA-1; SIGNAL TRANSDUCTION; SPIRO COMPOUNDS; UNCOUPLING AGENTS; VASODILATOR AGENTS;

EID: 4043087772     PISSN: 00097322     EISSN: None     Source Type: Journal    
DOI: 10.1161/01.CIR.0000126294.81407.7D     Document Type: Article

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