Preconditioning involves selective mitophagy mediated by parkin and p62/SQSTM1

PLoS ONE

Volumn 6, Issue 6, 2011, Pages

  Huang, Chengqun ^a   Andres, Allen M ^a   Ratliff, Eric P ^a   Hernandez, Genaro ^a   Lee, Pamela ^a   Gottlieb, Roberta A ^a  

^a SAN DIEGO STATE UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

AUTOPHAGY PROTEIN 5; PARKIN; PROTEIN P62; HEAT SHOCK PROTEIN; SIGNAL TRANSDUCING ADAPTOR PROTEIN; SQSTM1 PROTEIN, MOUSE; UBIQUITIN PROTEIN LIGASE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; AUTOPHAGY; CELL DEATH; CONTROLLED STUDY; DISORDERS OF MITOCHONDRIAL FUNCTIONS; EX VIVO STUDY; HEART DEPOLARIZATION; HEART INFARCTION PREVENTION; HEART MITOCHONDRION; HEART MUSCLE ISCHEMIA; HEART PROTECTION; IN VIVO STUDY; INTRACELLULAR TRANSPORT; ISOLATED HEART; MITOCHONDRIAL MEMBRANE POTENTIAL; MOUSE; NONHUMAN; PROTEIN DEPLETION; PROTEIN FUNCTION; PROTEIN TRANSPORT; UBIQUITINATION; WILD TYPE; ANIMAL; CELL LINE; CELL POLARITY; HEART MUSCLE; HEART MUSCLE CELL; METABOLISM; MITOCHONDRION; PATHOLOGY; PATHOPHYSIOLOGY; PHYSIOLOGICAL STRESS; REPERFUSION INJURY;

MUS; RATTUS;

ADAPTOR PROTEINS, SIGNAL TRANSDUCING; ANIMALS; AUTOPHAGY; CELL LINE; CELL POLARITY; HEAT-SHOCK PROTEINS; ISCHEMIC PRECONDITIONING, MYOCARDIAL; MICE; MITOCHONDRIA; MYOCARDIAL ISCHEMIA; MYOCARDIUM; MYOCYTES, CARDIAC; PROTEIN TRANSPORT; REPERFUSION INJURY; STRESS, PHYSIOLOGICAL; UBIQUITIN-PROTEIN LIGASES;

EID: 79958172986     PISSN: None     EISSN: 19326203     Source Type: Journal    
DOI: 10.1371/journal.pone.0020975     Document Type: Article

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