Aging Mouse Hearts Are Refractory to Infarct Size Reduction With Post-Conditioning

Journal of the American College of Cardiology

Volumn 51, Issue 14, 2008, Pages 1393-1398

  Przyklenk, Karin ^a   Maynard, Michelle ^a   Darling, Chad E ^a   Whittaker, Peter ^a,b  

^a UNIVERSITY OF MASSACHUSETTS MEDICAL SCHOOL   (United States)

^b WAYNE STATE UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

MITOGEN ACTIVATED PROTEIN KINASE 1; MITOGEN ACTIVATED PROTEIN KINASE 3; MITOGEN ACTIVATED PROTEIN KINASE PHOSPHATASE 1; PHOSPHATIDYLINOSITOL 3 KINASE; PROTEIN KINASE B;

AGING; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONDITIONING; CONTROLLED STUDY; ENZYME PHOSPHORYLATION; HEART INFARCTION SIZE; HEART MUSCLE ISCHEMIA; HEART MUSCLE REPERFUSION; HEART PROTECTION; MOUSE; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; PROTEIN DEPHOSPHORYLATION; PROTEIN EXPRESSION; UPREGULATION;

AGING; ANIMALS; HEART VENTRICLES; HYPERTROPHY, LEFT VENTRICULAR; MAP KINASE SIGNALING SYSTEM; MICE; MICE, INBRED C57BL; MYOCARDIAL INFARCTION; MYOCARDIAL REPERFUSION; MYOCARDIUM; PHENOTYPE; RANDOM ALLOCATION; TIME FACTORS; UP-REGULATION;

EID: 41149127345     PISSN: 07351097     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.jacc.2007.11.070     Document Type: Article

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