-
1
-
-
0037623908
-
Inhibition of myocardial injury by ischemic postconditioning during reperfusion: comparison with ischemic preconditioning
-
Zhao Z.Q., Corvera J.S., Halkos M.E., Kerendi F., Wang N.P., Guyton R.A., and Vinten-Johansen J. Inhibition of myocardial injury by ischemic postconditioning during reperfusion: comparison with ischemic preconditioning. Am. J. Physiol. Heart Circ. Physiol. 285 (2003) H579-H588
-
(2003)
Am. J. Physiol. Heart Circ. Physiol.
, vol.285
-
-
Zhao, Z.Q.1
Corvera, J.S.2
Halkos, M.E.3
Kerendi, F.4
Wang, N.P.5
Guyton, R.A.6
Vinten-Johansen, J.7
-
2
-
-
4043060632
-
Postconditioning: a form of "modified reperfusion" protects themyocardium by activating the phosphatidylinositol 3-kinase-Akt pathway
-
Tsang A., Hausenloy D.J., Mocanu M.M., and Yellon D.M. Postconditioning: a form of "modified reperfusion" protects themyocardium by activating the phosphatidylinositol 3-kinase-Akt pathway. Circ. Res. 95 (2004) 230-232
-
(2004)
Circ. Res.
, vol.95
, pp. 230-232
-
-
Tsang, A.1
Hausenloy, D.J.2
Mocanu, M.M.3
Yellon, D.M.4
-
3
-
-
3242690160
-
Multiple, brief coronary occlusions during early reperfusion protect rabbit hearts by targeting cell signaling pathways
-
Yang X.M., Proctor J.B., Cui L., Krieg T., Downey J.M., and Cohen M.V. Multiple, brief coronary occlusions during early reperfusion protect rabbit hearts by targeting cell signaling pathways. J. Am. Coll. Cardiol. 44 (2004) 1103-1110
-
(2004)
J. Am. Coll. Cardiol.
, vol.44
, pp. 1103-1110
-
-
Yang, X.M.1
Proctor, J.B.2
Cui, L.3
Krieg, T.4
Downey, J.M.5
Cohen, M.V.6
-
4
-
-
33646031836
-
Postconditioning protects rabbit hearts through a protein kinase C adenosine A2b receptor cascade
-
Philipp S., Yang X.M., Cui L., Davis A.M., Downey J.M., and Cohen M.V. Postconditioning protects rabbit hearts through a protein kinase C adenosine A2b receptor cascade. Cardiovasc. Res. 70 (2006) 308-314
-
(2006)
Cardiovasc. Res.
, vol.70
, pp. 308-314
-
-
Philipp, S.1
Yang, X.M.2
Cui, L.3
Davis, A.M.4
Downey, J.M.5
Cohen, M.V.6
-
5
-
-
32844463721
-
Post-conditioning reduces infarct size in the isolated rat heart: role of coronary flow and pressure and the nitric oxide/cGMP pathway, Basic Res
-
Penna C., Cappello S., Mancardi D., Raimondo S., Rastaldo R., Gattullo D., Losano G., and Pagliaro P. Post-conditioning reduces infarct size in the isolated rat heart: role of coronary flow and pressure and the nitric oxide/cGMP pathway, Basic Res. Cardiol 101 (2006) 168-179
-
(2006)
Cardiol
, vol.101
, pp. 168-179
-
-
Penna, C.1
Cappello, S.2
Mancardi, D.3
Raimondo, S.4
Rastaldo, R.5
Gattullo, D.6
Losano, G.7
Pagliaro, P.8
-
6
-
-
32844460892
-
Post-conditioning induced cardioprotection requires signaling through a redox-sensitive mechanism, mitochondrial ATP-sensitive K(+) channel and protein kinase C activation, Basic Res
-
Penna C., Rastaldo R., Mancardi D., Raimondo S., Cappello S., Gattullo D., Losano G., and Pagliaro P. Post-conditioning induced cardioprotection requires signaling through a redox-sensitive mechanism, mitochondrial ATP-sensitive K(+) channel and protein kinase C activation, Basic Res. Cardiol 101 (2006) 180-189
-
(2006)
Cardiol
, vol.101
, pp. 180-189
-
-
Penna, C.1
Rastaldo, R.2
Mancardi, D.3
Raimondo, S.4
Cappello, S.5
Gattullo, D.6
Losano, G.7
Pagliaro, P.8
-
7
-
-
29144474535
-
PI3-kinase regulates the mitochondrial transition pore in controlled reperfusion and postconditioning
-
Bopassa J.C., Ferrera R., Gateau-Roesch O., Couture-Lepetit E., and Ovize M. PI3-kinase regulates the mitochondrial transition pore in controlled reperfusion and postconditioning. Cardiovasc. Res. 69 (2006) 178-185
-
(2006)
Cardiovasc. Res.
, vol.69
, pp. 178-185
-
-
Bopassa, J.C.1
Ferrera, R.2
Gateau-Roesch, O.3
Couture-Lepetit, E.4
Ovize, M.5
-
8
-
-
25444480220
-
Postconditioning via stuttering reperfusion limits myocardial infarct size in rabbit hearts: role of ERK1/2
-
Jiang R., Maynard M., Whittaker P., Vinten-Johansen J., and Przyklenk K. Postconditioning via stuttering reperfusion limits myocardial infarct size in rabbit hearts: role of ERK1/2. Am. J. Physiol. Heart Circ. Physiol. 289 (2005) H1618-H1626
-
(2005)
Am. J. Physiol. Heart Circ. Physiol.
, vol.289
-
-
Jiang, R.1
Maynard, M.2
Whittaker, P.3
Vinten-Johansen, J.4
Przyklenk, K.5
-
9
-
-
33646016843
-
Infarct sparing effect of myocardial postconditioning is dependent on protein kinase C signaling
-
Zatta A.J., Kin H., Lee G., Wang N., Jiang R., Lust R., Reeves J.G., Mykytenko J., Guyton R.A., Zhao Z.Q., and Vinten-Johansen J. Infarct sparing effect of myocardial postconditioning is dependent on protein kinase C signaling. Cardiovasc. Res. 70 (2006) 315-324
-
(2006)
Cardiovasc. Res.
, vol.70
, pp. 315-324
-
-
Zatta, A.J.1
Kin, H.2
Lee, G.3
Wang, N.4
Jiang, R.5
Lust, R.6
Reeves, J.G.7
Mykytenko, J.8
Guyton, R.A.9
Zhao, Z.Q.10
Vinten-Johansen, J.11
-
10
-
-
38149141469
-
Increased mitochondrial calcium coexists with decreased reperfusion injury in postconditioned (but not preconditioned) hearts
-
Argaud L., Gateau-Roesch O., Augeul L., Couture-Lepetit E., Loufouat J., Gomez L., Robert D., and Ovize M. Increased mitochondrial calcium coexists with decreased reperfusion injury in postconditioned (but not preconditioned) hearts. Am. J. Physiol. Heart Circ. Physiol. 294 (2008) H386-H391
-
(2008)
Am. J. Physiol. Heart Circ. Physiol.
, vol.294
-
-
Argaud, L.1
Gateau-Roesch, O.2
Augeul, L.3
Couture-Lepetit, E.4
Loufouat, J.5
Gomez, L.6
Robert, D.7
Ovize, M.8
-
12
-
-
33745242990
-
No loss of cardioprotection by postconditioning in connexin 43-deficient mice
-
Heusch G., Büchert A., Feldhaus S., and Schulz R. No loss of cardioprotection by postconditioning in connexin 43-deficient mice. Basic Res. Cardiol. 101 (2006) 354-356
-
(2006)
Basic Res. Cardiol.
, vol.101
, pp. 354-356
-
-
Heusch, G.1
Büchert, A.2
Feldhaus, S.3
Schulz, R.4
-
13
-
-
35848941299
-
Connexin 43 in ischemic pre- and postconditioning
-
Schulz R., Boengler K., Totzeck A., Luo Y., Garcia-Dorado D., and Heusch G. Connexin 43 in ischemic pre- and postconditioning. Heart Fail. Rev. 12 (2007) 261-266
-
(2007)
Heart Fail. Rev.
, vol.12
, pp. 261-266
-
-
Schulz, R.1
Boengler, K.2
Totzeck, A.3
Luo, Y.4
Garcia-Dorado, D.5
Heusch, G.6
-
14
-
-
0842281645
-
Cell death: critical control points
-
Danial N.N., and Korsmeyer S.J. Cell death: critical control points. Cell 116 (2004) 205-219
-
(2004)
Cell
, vol.116
, pp. 205-219
-
-
Danial, N.N.1
Korsmeyer, S.J.2
-
15
-
-
8844235617
-
The mitochondrial death pathway and cardiac myocyte apoptosis
-
Crow M.T., Mani K., Nam Y.J., and Kitsis R.N. The mitochondrial death pathway and cardiac myocyte apoptosis. Circ. Res. 95 (2004) 957-970
-
(2004)
Circ. Res.
, vol.95
, pp. 957-970
-
-
Crow, M.T.1
Mani, K.2
Nam, Y.J.3
Kitsis, R.N.4
-
16
-
-
0036852691
-
Inhibition of apoptotic responses after ischemic stress in isolated hearts and cardiomyocytes
-
Hofstaetter B., Taimor G., Inserte J., Garcia-Dorado D., and Piper H.M. Inhibition of apoptotic responses after ischemic stress in isolated hearts and cardiomyocytes. Basic Res. Cardiol. 97 (2002) 479-488
-
(2002)
Basic Res. Cardiol.
, vol.97
, pp. 479-488
-
-
Hofstaetter, B.1
Taimor, G.2
Inserte, J.3
Garcia-Dorado, D.4
Piper, H.M.5
-
17
-
-
33747437290
-
Postconditioning attenuates cardiomyocyte apoptosis via inhibition of JNK and p38 mitogen-activated protein kinase signaling pathways
-
Sun H.Y., Wang N.P., Halkos M., Kerendi F., Kin H., Guyton R.A., Vinten-Johansen J., and Zhao Z.Q. Postconditioning attenuates cardiomyocyte apoptosis via inhibition of JNK and p38 mitogen-activated protein kinase signaling pathways. Apoptosis 11 (2006) 1583-1593
-
(2006)
Apoptosis
, vol.11
, pp. 1583-1593
-
-
Sun, H.Y.1
Wang, N.P.2
Halkos, M.3
Kerendi, F.4
Kin, H.5
Guyton, R.A.6
Vinten-Johansen, J.7
Zhao, Z.Q.8
-
18
-
-
33947373973
-
Preconditioning: the mitochondrial connection
-
Murphy E., and Steenbergen C. Preconditioning: the mitochondrial connection. Ann. Rev. Physiol. 69 (2007) 51-67
-
(2007)
Ann. Rev. Physiol.
, vol.69
, pp. 51-67
-
-
Murphy, E.1
Steenbergen, C.2
-
19
-
-
0141751749
-
Preconditioning the myocardium: from cellular physiology to clinical cardiology
-
Yellon D.M., and Downey J.M. Preconditioning the myocardium: from cellular physiology to clinical cardiology. Physiol. Rev. 83 (2003) 1113-1151
-
(2003)
Physiol. Rev.
, vol.83
, pp. 1113-1151
-
-
Yellon, D.M.1
Downey, J.M.2
-
20
-
-
36849037512
-
Pim-1 regulates cardiomyocyte survival downstream of Akt
-
Muraski J.A., Rota M., Misao Y., Fransioli J., Cottage C., Gude N., Esposito G., Delucchi F., Arcarese M., Alvarez R., Siddiqi S., Emmanuel G.N., Wu W., Fischer K., Martindale J.J., Glembotski C.C., Leri A., Kajstura J., Magnuson N., Berns A., Beretta R.M., Houser S.R., Schaefer E.M., Anversa P., and Sussman M.A. Pim-1 regulates cardiomyocyte survival downstream of Akt. Nat. Med. 13 (2007) 1467-1475
-
(2007)
Nat. Med.
, vol.13
, pp. 1467-1475
-
-
Muraski, J.A.1
Rota, M.2
Misao, Y.3
Fransioli, J.4
Cottage, C.5
Gude, N.6
Esposito, G.7
Delucchi, F.8
Arcarese, M.9
Alvarez, R.10
Siddiqi, S.11
Emmanuel, G.N.12
Wu, W.13
Fischer, K.14
Martindale, J.J.15
Glembotski, C.C.16
Leri, A.17
Kajstura, J.18
Magnuson, N.19
Berns, A.20
Beretta, R.M.21
Houser, S.R.22
Schaefer, E.M.23
Anversa, P.24
Sussman, M.A.25
more..
-
21
-
-
0030609009
-
Enforced expression of the Mr 33,000 Pim-1 kinase enhances factor-independent survival and inhibits apoptosis in murine myeloid cells
-
Lilly M., and Kraft A. Enforced expression of the Mr 33,000 Pim-1 kinase enhances factor-independent survival and inhibits apoptosis in murine myeloid cells. Cancer Res. 57 (1997) 5348-5355
-
(1997)
Cancer Res.
, vol.57
, pp. 5348-5355
-
-
Lilly, M.1
Kraft, A.2
-
22
-
-
0030890933
-
Transduction of interleukin-2 antiapoptotic and proliferative signals via Akt protein kinase
-
Ahmed N.N., Grimes H.L., Bellacosa A., Chan T.O., and Tsichlis P.N. Transduction of interleukin-2 antiapoptotic and proliferative signals via Akt protein kinase. Proc. Natl. Acad. Sci. U. S. A. 94 (1997) 3627-3632
-
(1997)
Proc. Natl. Acad. Sci. U. S. A.
, vol.94
, pp. 3627-3632
-
-
Ahmed, N.N.1
Grimes, H.L.2
Bellacosa, A.3
Chan, T.O.4
Tsichlis, P.N.5
-
23
-
-
26444448463
-
The survival kinases Akt and Pim as potential pharmacological targets
-
Amaravadi R., and Thompson C.B. The survival kinases Akt and Pim as potential pharmacological targets. J. Clin. Invest. 115 (2005) 2618-2624
-
(2005)
J. Clin. Invest.
, vol.115
, pp. 2618-2624
-
-
Amaravadi, R.1
Thompson, C.B.2
-
24
-
-
46149089709
-
Postconditioning and intermittent bradykinin induced cardioprotection require cyclooxygenase activation and prostacyclin release during reperfusion
-
Penna C., Mancardi D., Tullio F., and Pagliaro P. Postconditioning and intermittent bradykinin induced cardioprotection require cyclooxygenase activation and prostacyclin release during reperfusion. Basic Res. Cardiol. 103 (2008) 368-377
-
(2008)
Basic Res. Cardiol.
, vol.103
, pp. 368-377
-
-
Penna, C.1
Mancardi, D.2
Tullio, F.3
Pagliaro, P.4
-
25
-
-
0035903235
-
Bid is cleaved by calpain to an active fragment in vitro and during myocardial ischemia/reperfusion
-
Chen M., He H., Zhan S., Krajewski S., Reed J.C., and Gottlieb R.A. Bid is cleaved by calpain to an active fragment in vitro and during myocardial ischemia/reperfusion. J. Biol. Chem. 276 (2001) 30724-30728
-
(2001)
J. Biol. Chem.
, vol.276
, pp. 30724-30728
-
-
Chen, M.1
He, H.2
Zhan, S.3
Krajewski, S.4
Reed, J.C.5
Gottlieb, R.A.6
-
26
-
-
34147188924
-
Loss of ischemic preconditioning's cardioprotection in aged mouse hearts is associated with reduced gap junctional and mitochondrial levels of connexin 43
-
Boengler K., Konietzka I., Buechert A., Heinen Y., Garcia-Dorado D., Heusch G., and Schulz R. Loss of ischemic preconditioning's cardioprotection in aged mouse hearts is associated with reduced gap junctional and mitochondrial levels of connexin 43. Am. J. Physiol. Heart Circ. Physiol. 292 (2007) H1764-H1769
-
(2007)
Am. J. Physiol. Heart Circ. Physiol.
, vol.292
-
-
Boengler, K.1
Konietzka, I.2
Buechert, A.3
Heinen, Y.4
Garcia-Dorado, D.5
Heusch, G.6
Schulz, R.7
-
27
-
-
0346727168
-
2+-induced mitochondrial permeability transition
-
2+-induced mitochondrial permeability transition. Cardiovasc Res. 61 (2004) 115-122
-
(2004)
Cardiovasc Res.
, vol.61
, pp. 115-122
-
-
Argaud, L.1
Gateau-Roesch, O.2
Chalabreysse, L.3
Gomez, L.4
Loufouat, J.5
Thivolet-Béjui, F.6
Robert, D.7
Ovize, M.8
-
28
-
-
0017184389
-
A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding
-
Bradford M.M. A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding. Anal. Biochem. 7 (1976) 248-254
-
(1976)
Anal. Biochem.
, vol.7
, pp. 248-254
-
-
Bradford, M.M.1
-
29
-
-
0033536203
-
The PIM-1 serine kinase prolongs survival and inhibits apoptosis-related mitochondrial dysfunction in part through a bcl-2-dependent pathway
-
Lilly M., Sandholm J., Cooper J.J., Koskinen P.J., and Kraft A. The PIM-1 serine kinase prolongs survival and inhibits apoptosis-related mitochondrial dysfunction in part through a bcl-2-dependent pathway. Oncogene 18 (1999) 4022-4031
-
(1999)
Oncogene
, vol.18
, pp. 4022-4031
-
-
Lilly, M.1
Sandholm, J.2
Cooper, J.J.3
Koskinen, P.J.4
Kraft, A.5
-
30
-
-
85047692700
-
Glycogen synthase kinase-3beta mediates convergence of protection signaling to inhibit the mitochondrial permeability transition pore
-
Juhaszova M., Zorov D.B., Kim S.H., Pepe S., Fu Q., Fishbein K.W., Ziman B.D., Wang S., Ytrehus K., Antos C.L., Olson E.N., and Sollott S.J. Glycogen synthase kinase-3beta mediates convergence of protection signaling to inhibit the mitochondrial permeability transition pore. J. Clin. Invest. 113 (2004) 1535-1549
-
(2004)
J. Clin. Invest.
, vol.113
, pp. 1535-1549
-
-
Juhaszova, M.1
Zorov, D.B.2
Kim, S.H.3
Pepe, S.4
Fu, Q.5
Fishbein, K.W.6
Ziman, B.D.7
Wang, S.8
Ytrehus, K.9
Antos, C.L.10
Olson, E.N.11
Sollott, S.J.12
-
31
-
-
21244500204
-
Inhibition of GSK-3beta as a target for cardioprotection: the importance of timing, location, duration and degree of inhibition
-
Murphy E., and Steenbergen C. Inhibition of GSK-3beta as a target for cardioprotection: the importance of timing, location, duration and degree of inhibition. Expert Opin. Ther. Targets 9 (2005) 447-456
-
(2005)
Expert Opin. Ther. Targets
, vol.9
, pp. 447-456
-
-
Murphy, E.1
Steenbergen, C.2
-
32
-
-
2942737344
-
Inhibit GSK-3beta or there's heartbreak dead ahead
-
Murphy E. Inhibit GSK-3beta or there's heartbreak dead ahead. J. Clin. Invest 113 (2004) 1526-1528
-
(2004)
J. Clin. Invest
, vol.113
, pp. 1526-1528
-
-
Murphy, E.1
-
33
-
-
44449133771
-
Inhibition of GSK3beta by postconditioning is required to prevent opening of the mitochondrial permeability transition pore during reperfusion
-
Gomez L., Paillard M., Thibault H., Derumeaux G., and Ovize M. Inhibition of GSK3beta by postconditioning is required to prevent opening of the mitochondrial permeability transition pore during reperfusion. Circulation 117 (2008) 2761-2768
-
(2008)
Circulation
, vol.117
, pp. 2761-2768
-
-
Gomez, L.1
Paillard, M.2
Thibault, H.3
Derumeaux, G.4
Ovize, M.5
-
34
-
-
19944373389
-
Nicastrin, presenilin, APH-1, and PEN-2 form active gamma-secretase complexes in mitochondria
-
Hansson C.A., Frykman S., Farmery M.R., Tjernberg L.O., Nilsberth C., Pursglove S.E., Ito A., Winblad B., Cowburn R.F., Thyberg J., and Ankarcrona M. Nicastrin, presenilin, APH-1, and PEN-2 form active gamma-secretase complexes in mitochondria. J. Biol. Chem. 279 (2004) 51654-51660
-
(2004)
J. Biol. Chem.
, vol.279
, pp. 51654-51660
-
-
Hansson, C.A.1
Frykman, S.2
Farmery, M.R.3
Tjernberg, L.O.4
Nilsberth, C.5
Pursglove, S.E.6
Ito, A.7
Winblad, B.8
Cowburn, R.F.9
Thyberg, J.10
Ankarcrona, M.11
-
35
-
-
38849201088
-
Mitochondrial connexin43 as a new player in the pathophysiology of myocardial ischaemia-reperfusion injury
-
Ruiz-Meana M., Rodríguez-Sinovas A., Cabestrero A., Boengler K., Heusch G., and Garcia-Dorado D. Mitochondrial connexin43 as a new player in the pathophysiology of myocardial ischaemia-reperfusion injury. Cardiovasc. Res. 77 (2008) 325-333
-
(2008)
Cardiovasc. Res.
, vol.77
, pp. 325-333
-
-
Ruiz-Meana, M.1
Rodríguez-Sinovas, A.2
Cabestrero, A.3
Boengler, K.4
Heusch, G.5
Garcia-Dorado, D.6
-
36
-
-
34248682977
-
Mitochondrial translocation of p53 underlies the selective death of hippocampal CA1 neurons after global cerebral ischaemia
-
Endo H., Saito A., and Chan P.H. Mitochondrial translocation of p53 underlies the selective death of hippocampal CA1 neurons after global cerebral ischaemia. Biochem. Soc. Trans. 34 (2006) 1283-1286
-
(2006)
Biochem. Soc. Trans.
, vol.34
, pp. 1283-1286
-
-
Endo, H.1
Saito, A.2
Chan, P.H.3
-
37
-
-
34547110113
-
Mitochondria transmit apoptosis signalling in cardiomyocyte-like cells and isolated hearts exposed to experimental ischemia-reperfusion injury
-
Neuzil J., Widén C., Gellert N., Swettenham E., Zobalova R., Dong L.F., Wang X.F., Lidebjer C., Dalen H., Headrick J.P., and Witting P.K. Mitochondria transmit apoptosis signalling in cardiomyocyte-like cells and isolated hearts exposed to experimental ischemia-reperfusion injury. Redox Rep. 12 (2007) 148-162
-
(2007)
Redox Rep.
, vol.12
, pp. 148-162
-
-
Neuzil, J.1
Widén, C.2
Gellert, N.3
Swettenham, E.4
Zobalova, R.5
Dong, L.F.6
Wang, X.F.7
Lidebjer, C.8
Dalen, H.9
Headrick, J.P.10
Witting, P.K.11
-
38
-
-
0030870555
-
The bcl-2 gene product prevents programmed cell death of ventricular myocytes
-
Kirshenbaum L.A., and de Moissac D. The bcl-2 gene product prevents programmed cell death of ventricular myocytes. Circulation 96 (1997) 1580-1585
-
(1997)
Circulation
, vol.96
, pp. 1580-1585
-
-
Kirshenbaum, L.A.1
de Moissac, D.2
-
39
-
-
0035695645
-
Regulation of sodium-calcium exchange and mitochondrial energetics by Bcl-2 in the heart of transgenic mice
-
Zhu L., Yu Y., Chua B.H., Ho Y.S., and Kuo T.H. Regulation of sodium-calcium exchange and mitochondrial energetics by Bcl-2 in the heart of transgenic mice. J. Mol. Cell Cardiol. 33 (2001) 2135-2144
-
(2001)
J. Mol. Cell Cardiol.
, vol.33
, pp. 2135-2144
-
-
Zhu, L.1
Yu, Y.2
Chua, B.H.3
Ho, Y.S.4
Kuo, T.H.5
-
40
-
-
0034698118
-
Morphological and molecular characterization of adult cardiomyocyte apoptosis during hypoxia and reoxygenation
-
Kang P.M., Haunstetter A., Aoki H., Usheva A., and Izumo S. Morphological and molecular characterization of adult cardiomyocyte apoptosis during hypoxia and reoxygenation. Circ. Res. 87 (2000) 118-125
-
(2000)
Circ. Res.
, vol.87
, pp. 118-125
-
-
Kang, P.M.1
Haunstetter, A.2
Aoki, H.3
Usheva, A.4
Izumo, S.5
-
41
-
-
42349105567
-
The paradigm of postconditioning to protect the heart
-
Penna C., Mancardi D., Raimondo S., Geuna S., and Pagliaro P. The paradigm of postconditioning to protect the heart. J. Cell Mol. Med. 12 (2008) 435-458
-
(2008)
J. Cell Mol. Med.
, vol.12
, pp. 435-458
-
-
Penna, C.1
Mancardi, D.2
Raimondo, S.3
Geuna, S.4
Pagliaro, P.5
-
42
-
-
59649114814
-
Ischemic postconditioning in pigs no causal role for RISK activation
-
Skyschally A., van Caster P., Boengler K., Gres P., Musiolik J., Schilawa D., Schulz R., and Heusch G. Ischemic postconditioning in pigs no causal role for RISK activation. Circ. Res. 104 (2008) 15-18
-
(2008)
Circ. Res.
, vol.104
, pp. 15-18
-
-
Skyschally, A.1
van Caster, P.2
Boengler, K.3
Gres, P.4
Musiolik, J.5
Schilawa, D.6
Schulz, R.7
Heusch, G.8
-
43
-
-
0037799220
-
Protein phosphatase 2A regulates the stability of Pim protein kinases
-
Losman J.A., Chen X.P., Vuong B.Q., Fay S., and Rothman P.B. Protein phosphatase 2A regulates the stability of Pim protein kinases. J. Biol. Chem. 278 (2003) 4800-4805
-
(2003)
J. Biol. Chem.
, vol.278
, pp. 4800-4805
-
-
Losman, J.A.1
Chen, X.P.2
Vuong, B.Q.3
Fay, S.4
Rothman, P.B.5
-
44
-
-
0036350661
-
Role of protein phosphatases in hypoxic preconditioning
-
Ladilov Y., Maxeiner H., Wolf C., Schäfer C., Meuter K., and Piper H.M. Role of protein phosphatases in hypoxic preconditioning. Am. J. Physiol. Heart Circ. Physiol. 283 (2002) H1092-H1098
-
(2002)
Am. J. Physiol. Heart Circ. Physiol.
, vol.283
-
-
Ladilov, Y.1
Maxeiner, H.2
Wolf, C.3
Schäfer, C.4
Meuter, K.5
Piper, H.M.6
-
45
-
-
26444551639
-
Inhibition of phosphatase activity enhances preconditioning and limits cell death in the ischemic/reperfused aged rat heart
-
Fenton R.A., Dickson E.W., and Dobson Jr. J.G. Inhibition of phosphatase activity enhances preconditioning and limits cell death in the ischemic/reperfused aged rat heart. Life Sci. 77 (2005) 3375-3388
-
(2005)
Life Sci.
, vol.77
, pp. 3375-3388
-
-
Fenton, R.A.1
Dickson, E.W.2
Dobson Jr., J.G.3
-
46
-
-
27644560497
-
Isoflurane postconditioning prevents opening of the mitochondrial permeability transition pore through inhibition of glycogen synthase kinase 3beta
-
Feng J., Lucchinetti E., Ahuja P., Pasch T., Perriard J.C., and Zaugg M. Isoflurane postconditioning prevents opening of the mitochondrial permeability transition pore through inhibition of glycogen synthase kinase 3beta. Anesthesiology 103 (2005) 987-995
-
(2005)
Anesthesiology
, vol.103
, pp. 987-995
-
-
Feng, J.1
Lucchinetti, E.2
Ahuja, P.3
Pasch, T.4
Perriard, J.C.5
Zaugg, M.6
-
47
-
-
51749092348
-
Glycogen synthase kinase-3 inactivation is not required for ischemic preconditioning or postconditioning in the mouse
-
Nishino Y., Webb I.G., Davidson S.M., Ahmed A.I., Clark J.E., Jacquet S., Shah A.M., Miura T., Yellon D.M., Avkiran M., and Marber M.S. Glycogen synthase kinase-3 inactivation is not required for ischemic preconditioning or postconditioning in the mouse. Circ. Res. 103 (2008) 307-314
-
(2008)
Circ. Res.
, vol.103
, pp. 307-314
-
-
Nishino, Y.1
Webb, I.G.2
Davidson, S.M.3
Ahmed, A.I.4
Clark, J.E.5
Jacquet, S.6
Shah, A.M.7
Miura, T.8
Yellon, D.M.9
Avkiran, M.10
Marber, M.S.11
-
48
-
-
7744227096
-
Glycogen synthase kinase-3beta phosphorylates Bax and promotes its mitochondrial localization during neuronal apoptosis
-
Linseman D.A., Butts B.D., Precht T.A., Phelps R.A., Le S.S., Laessig T.A., Bouchard R.J., Florez-McClure M.L., and Heidenreich K.A. Glycogen synthase kinase-3beta phosphorylates Bax and promotes its mitochondrial localization during neuronal apoptosis. J. Neurosci. 24 (2004) 9993-10002
-
(2004)
J. Neurosci.
, vol.24
, pp. 9993-10002
-
-
Linseman, D.A.1
Butts, B.D.2
Precht, T.A.3
Phelps, R.A.4
Le, S.S.5
Laessig, T.A.6
Bouchard, R.J.7
Florez-McClure, M.L.8
Heidenreich, K.A.9
-
49
-
-
33751192473
-
What are caspases 3 and 7 doing upstream of the mitochondria?
-
Kuribayashi K., Mayes P.A., and El-Deiry W.S. What are caspases 3 and 7 doing upstream of the mitochondria?. Cancer Biol. Ther. 5 (2006) 763-765
-
(2006)
Cancer Biol. Ther.
, vol.5
, pp. 763-765
-
-
Kuribayashi, K.1
Mayes, P.A.2
El-Deiry, W.S.3
-
50
-
-
33745813297
-
Connexin 43 signalling and cardioprotection
-
Boengler K., Schulz R., and Heusch G. Connexin 43 signalling and cardioprotection. Heart 92 (2006) 1724-1727
-
(2006)
Heart
, vol.92
, pp. 1724-1727
-
-
Boengler, K.1
Schulz, R.2
Heusch, G.3
-
51
-
-
25444512382
-
Impairment of diazoxide-induced formation of reactive oxygen species and loss of cardioprotection in connexin 43 deficient mice
-
Heinzel F.R., Luo Y., Li X., Boengler K., Buechert A., García-Dorado D., Di Lisa F., Schulz R., and Heusch G. Impairment of diazoxide-induced formation of reactive oxygen species and loss of cardioprotection in connexin 43 deficient mice. Circ. Res. 97 (2005) 583-586
-
(2005)
Circ. Res.
, vol.97
, pp. 583-586
-
-
Heinzel, F.R.1
Luo, Y.2
Li, X.3
Boengler, K.4
Buechert, A.5
García-Dorado, D.6
Di Lisa, F.7
Schulz, R.8
Heusch, G.9
-
52
-
-
21744434924
-
Connexin 43 in cardiomyocyte mitochondria and its increase by ischemic preconditioning
-
Boengler K., Dodoni G., Rodriguez-Sinovas A., Cabestrero A., Ruiz-Meana M., Gres P., Konietzka I., Lopez-Iglesias C., Garcia-Dorado D., Di Lisa F., Heusch G., and Schulz R. Connexin 43 in cardiomyocyte mitochondria and its increase by ischemic preconditioning. Cardiovasc. Res. 67 (2005) 234-244
-
(2005)
Cardiovasc. Res.
, vol.67
, pp. 234-244
-
-
Boengler, K.1
Dodoni, G.2
Rodriguez-Sinovas, A.3
Cabestrero, A.4
Ruiz-Meana, M.5
Gres, P.6
Konietzka, I.7
Lopez-Iglesias, C.8
Garcia-Dorado, D.9
Di Lisa, F.10
Heusch, G.11
Schulz, R.12
-
53
-
-
34848840565
-
Reactive oxygen species trigger ischemic and pharmacological postconditioning: in vivo and in vitro characterization
-
Tsutsumi Y.M., Yokoyama T., Horikawa Y., Roth D.M., and Patel H.H. Reactive oxygen species trigger ischemic and pharmacological postconditioning: in vivo and in vitro characterization. Life Sci. 81 (2007) 1223-1227
-
(2007)
Life Sci.
, vol.81
, pp. 1223-1227
-
-
Tsutsumi, Y.M.1
Yokoyama, T.2
Horikawa, Y.3
Roth, D.M.4
Patel, H.H.5
-
54
-
-
34250004650
-
Intermittent activation of bradykinin B2 receptors and mitochondrial KATP channels trigger cardiac postconditioning through redox signaling
-
Penna C., Mancardi D., Rastaldo R., Losano G., and Pagliaro P. Intermittent activation of bradykinin B2 receptors and mitochondrial KATP channels trigger cardiac postconditioning through redox signaling. Cardiovasc. Res. 75 (2007) 168-177
-
(2007)
Cardiovasc. Res.
, vol.75
, pp. 168-177
-
-
Penna, C.1
Mancardi, D.2
Rastaldo, R.3
Losano, G.4
Pagliaro, P.5
-
55
-
-
33751409127
-
Cardiac mitochondrial connexin 43 regulates apoptosis
-
Goubaeva F., Mikami M., Giardina S., Ding B., Abe J., and Yang J. Cardiac mitochondrial connexin 43 regulates apoptosis. Biochem. Biophys. Res. Commun. 352 (2007) 97-103
-
(2007)
Biochem. Biophys. Res. Commun.
, vol.352
, pp. 97-103
-
-
Goubaeva, F.1
Mikami, M.2
Giardina, S.3
Ding, B.4
Abe, J.5
Yang, J.6
-
56
-
-
33746207799
-
Mitochondria and preconditioning: a connexin connection?
-
Halestrap A.P. Mitochondria and preconditioning: a connexin connection?. Circ. Res. 99 (2006) 10-12
-
(2006)
Circ. Res.
, vol.99
, pp. 10-12
-
-
Halestrap, A.P.1
|