Postconditioning inhibits mitochondrial permeability transition

Circulation

Volumn 111, Issue 2, 2005, Pages 194-197

  Argaud, Laurent ^a,b   Gateau Roesch, Odile ^a   Raisky, Olivier ^a   Loufouat, Joseph ^a   Robert, Dominique ^b   Ovize, Michel ^a,c  

^a UNIVERSITÉ LYON 1   (France)

^b HOSPICES CIVILS DE LYON   (France)

^c INSERM   (France)

Author keywords

Ischemia; Myocardial infarction; Reperfusion

Indexed keywords

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; HEART INFARCTION; HEART INFARCTION SIZE; HEART MUSCLE ISCHEMIA; HEART MUSCLE POSTCONDITIONING; HEART MUSCLE REPERFUSION; HEART PROTECTION; MALE; MEMBRANE PERMEABILITY; MITOCHONDRIAL MEMBRANE; NONHUMAN; PRIORITY JOURNAL; RABBIT; REPERFUSION INJURY;

ANIMALS; APOPTOSIS; CALCIUM; CORONARY DISEASE; CYCLOSPORINE; EXTRACELLULAR SIGNAL-REGULATED MAP KINASES; ION CHANNEL GATING; ION CHANNELS; ION TRANSPORT; MALE; MAP KINASE SIGNALING SYSTEM; MEMBRANE POTENTIALS; MITOCHONDRIA, HEART; MITOCHONDRIAL MEMBRANE TRANSPORT PROTEINS; MYOCARDIAL INFARCTION; MYOCARDIAL ISCHEMIA; MYOCARDIAL REPERFUSION INJURY; NECROSIS; OXIDATIVE STRESS; PROTEIN-SERINE-THREONINE KINASES; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS C-AKT; RABBITS; RANDOM ALLOCATION; TIME FACTORS;

EID: 12344317925     PISSN: 00097322     EISSN: None     Source Type: Journal    
DOI: 10.1161/01.CIR.0000151290.04952.3B     Document Type: Article

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