Apoptosome dysfunction in human cancer

Apoptosis

Volumn 9, Issue 6, 2004, Pages 691-704

  Hajra, K M ^a   Liu, J R ^a  

^a UNIVERSITY OF MICHIGAN MEDICAL SCHOOL   (United States)

Author keywords

Apaf 1; Apoptosis; Apoptosome; Bcl 2; IAP

Indexed keywords

ADENOVIRUS VECTOR; ANTISENSE OLIGONUCLEOTIDE; APOPTOSOME; APOPTOTIC PROTEASE ACTIVATING FACTOR 1; BH3 PROTEIN; CARBANILAMIDE DERIVATIVE; CASPASE 9; CYTOCHROME C; DEGUELIN; HEAT SHOCK PROTEIN; HEAT SHOCK PROTEIN 70; HEAT SHOCK PROTEIN 90; INHIBITOR OF APOPTOSIS PROTEIN; PERIFOSINE; PROTEIN BAK; PROTEIN BAX; PROTEIN BCL 2; PROTEIN BCL XL; PROTEIN KINASE INHIBITOR; PROTHYMOSIN ALPHA; SECOND MITOCHONDRIAL ACTIVATOR OF CASPASE; TUMOR NECROSIS FACTOR ALPHA; APAF1 PROTEIN, HUMAN; CASPASE; CYTOCHROME C''; PROTEIN; TUMOR NECROSIS FACTOR;

APOPTOSIS; ARTICLE; BREAST CANCER; CANCER; CANCER ADJUVANT THERAPY; CARCINOGENESIS; CELL COUNT; CELL DEATH; CELL GROWTH; CELL KILLING; CELL SURVIVAL; CLINICAL TRIAL; DNA DAMAGE; ENZYME ACTIVATION; ENZYME RELEASE; GENE THERAPY; HETEROZYGOSITY LOSS; HUMAN; HYPOXIA; MITOCHONDRION; NONHUMAN; PRIORITY JOURNAL; PROTEIN DEGRADATION; PROTEIN DOMAIN; PROTEIN EXPRESSION; PROTEIN FAMILY; PROTEIN FUNCTION; PROTEIN PHOSPHORYLATION; PROTEIN PROTEIN INTERACTION; SIGNAL TRANSDUCTION; SOLID TUMOR; UPREGULATION; ANIMAL; BIOLOGICAL MODEL; METABOLISM; NEOPLASM; PATHOLOGY; REVIEW; SECRETION;

ANIMALS; APOPTOSIS; APOPTOTIC PROTEASE-ACTIVATING FACTOR 1; CASPASES; CYTOCHROME C GROUP; ENZYME ACTIVATION; HUMANS; MITOCHONDRIA; MODELS, BIOLOGICAL; NEOPLASMS; PROTEINS; TUMOR NECROSIS FACTORS;

EID: 17444383660     PISSN: 13608185     EISSN: None     Source Type: Journal    
DOI: 10.1023/B:APPT.0000045786.98031.1d     Document Type: Article

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