The combined functions of proapoptotic Bcl-2 family members Bak and Bax are essential for normal development of multiple tissues

Molecular Cell

Volumn 6, Issue 6, 2000, Pages 1389-1399

  Lindsten, Tullia ^a   Ross, Andrea J ^b   King, Ayala ^a   Zong, Wei Xing ^a   Rathmell, Jeffrey C ^a   Shiels, Helena A ^c   Ulrich, Eugen ^d   Waymire, Katrina G ^b   Mahar, Patryce ^b   Frauwirth, Kenneth ^a   Chen, Yifeng ^a   Wei, Michael ^g   Eng, Vicki M ^e   Adelman, David M ^f   Simon, M Celeste ^f   Ma, Averil ^c   Golden, Jeffrey A ^a   Evan, Gerard ^d   Korsmeyer, Stanley J ^g   MacGregor, Grant R ^b   Thompson, Craig B ^a   more..

^a UNIVERSITY OF PENNSYLVANIA   (United States)

^b EMORY UNIVERSITY SCHOOL OF MEDICINE   (United States)

^c UNIVERSITY OF CHICAGO   (United States)

^d UNIVERSITY OF CALIFORNIA   (United States)

^e UNIVERSITY OF PENNSYLVANIA   (United States)

^f HOWARD HUGHES MEDICAL INSTITUTE   (United States)

^g DANA FARBER CANCER INSTITUTE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ETOPOSIDE; PROTEIN BAK; PROTEIN BAX; PROTEIN BCL 2;

ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CELL DEATH; HEMATOPOIETIC STEM CELL; KNOCKOUT MOUSE; MEMORY CELL; NONHUMAN; PHENOTYPE; RADIATION INJURY; SKULL DEFECT; VAGINA APLASIA;

ANIMALIA; MAMMALIA;

EID: 0034508217     PISSN: 10972765     EISSN: None     Source Type: Journal    
DOI: 10.1016/S1097-2765(00)00136-2     Document Type: Article

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