13 reasons why the brain is susceptible to oxidative stress

Redox Biology

Volumn 15, Issue , 2018, Pages 490-503

  Cobley, James Nathan ^a   Fiorello, Maria Luisa ^a   Bailey, Damian Miles ^b  

^a UNIVERSITY OF THE HIGHLANDS AND ISLANDS   (United Kingdom)

^b UNIVERSITY OF GLAMORGAN   (United Kingdom)

Author keywords

Brain; Mitochondria; Neurodegeneration; Oxidative stress; Redox signalling

Indexed keywords

CYTOCHROME C OXIDASE; DOPAMINE; GLUCOSE; GLUCOSYLCERAMIDASE; GLUTAMIC ACID; GLUTATHIONE; LONG UNTRANSLATED RNA; MICRORNA; PHOSPHOLIPID HYDROPEROXIDE GLUTATHIONE PEROXIDASE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE OXIDASE 2; SUPEROXIDE DISMUTASE; ANTIOXIDANT; FREE RADICAL; REACTIVE OXYGEN METABOLITE;

AMYOTROPHIC LATERAL SCLEROSIS; CALCIUM CELL LEVEL; CALCIUM SIGNALING; CALCIUM TRANSPORT; ENZYME ACTIVITY; FENTON REACTION; HIPPOCAMPUS; HUMAN; IN VIVO STUDY; LIPID PEROXIDATION; LONG TERM POTENTIATION; MICROGLIA; MITOCHONDRIAL PERMEABILITY; MITOCHONDRION; NERVE CELL PLASTICITY; NERVE DEGENERATION; NONHUMAN; OXIDATIVE STRESS; PARKINSON DISEASE; PI3K/AKT SIGNALING; PRIORITY JOURNAL; REVIEW; BRAIN; METABOLISM; NERVE CELL; PATHOLOGY; PHYSIOLOGY;

ANTIOXIDANTS; BRAIN; FREE RADICALS; HUMANS; MITOCHONDRIA; NEURONS; OXIDATIVE STRESS; REACTIVE OXYGEN SPECIES;

EID: 85041584254     PISSN: 22132317     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.redox.2018.01.008     Document Type: Review

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