The neurological phenotype of ataxia-telangiectasia: Solving a persistent puzzle

DNA Repair

Volumn 7, Issue 7, 2008, Pages 1028-1038

  Biton, Sharon ^a   Barzilai, Ari ^a   Shiloh, Yosef ^a  

^a TEL AVIV UNIVERSITY   (Israel)

Author keywords

Ataxia telangiectasia; ATM; DNA; DNA damage response; Double strand breaks

Indexed keywords

ATM PROTEIN;

ARTICLE; ATAXIA TELANGIECTASIA; CANCER SUSCEPTIBILITY; CELL PROLIFERATION; CELL SURVIVAL; CENTRAL NERVOUS SYSTEM; DNA DAMAGE; DNA REPAIR; DNA STRAND BREAKAGE; ENZYME INACTIVATION; GENOMIC INSTABILITY; HUMAN; IMMUNE DEFICIENCY; IONIZING RADIATION; NERVE CELL DEGENERATION; NERVE CELL NECROSIS; NERVOUS SYSTEM DEVELOPMENT; NEUROBIOLOGY; OXIDATIVE STRESS; PHOSPHORYLATION; PRIORITY JOURNAL; RADIOSENSITIVITY;

ANIMALS; ATAXIA TELANGIECTASIA; CELL CYCLE PROTEINS; DNA DAMAGE; DNA-BINDING PROTEINS; GENOMIC INSTABILITY; HUMANS; MICE; NEURONS; OXIDATIVE STRESS; PHENOTYPE; PROTEIN-SERINE-THREONINE KINASES; TUMOR SUPPRESSOR PROTEINS;

ATAXIA TELANGIECTASIA;

EID: 44949238447     PISSN: 15687864     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.dnarep.2008.03.006     Document Type: Article

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