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Volumn 20, Issue 3, 2001, Pages 289-294

Ataxia-telangiectasia: Chronic activation of damage-responsive functions is reduced by α-lipoic acid

(8) Gatei, Magtouf a Shkedy, Dganit b Khanna, Kum Kum a Uziel, Tamar b Shiloh, Yosef b Pandita, Tej K c Lavin, Martin F a,d Rotman, Galit b

a ROYAL BRISBANE AND WOMEN'S HOSPITAL (Australia)

b TEL AVIV UNIVERSITY (Israel)

c Columbia University ^* (United States)

d UNIVERSITY OF QUEENSLAND (Australia)

Author keywords

lipoic acid; Ataxia Telangiectasia; ATM; cdc2; Oxidative stress; p21WAF1 CIP1; p53

Indexed keywords

PROTEIN KINASE; PROTEIN P21; PROTEIN P53; REACTIVE OXYGEN METABOLITE; THIOCTIC ACID;

ARTICLE; ATAXIA TELANGIECTASIA; CONTROLLED STUDY; DNA DAMAGE; GENOTOXICITY; HUMAN; HUMAN CELL; IONIZING RADIATION; MACROMOLECULE; OXIDATIVE STRESS; PHENOTYPE; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION; RADIOSENSITIVITY;

ANTIOXIDANTS; ATAXIA TELANGIECTASIA; CASE-CONTROL STUDIES; CDC2 PROTEIN KINASE; CELLS, CULTURED; CYCLIN-DEPENDENT KINASE INHIBITOR P21; CYCLINS; CYCLOHEXIMIDE; DNA DAMAGE; HUMANS; OXIDATIVE STRESS; PHOSPHORYLATION; REACTIVE OXYGEN SPECIES; THIOCTIC ACID; TUMOR SUPPRESSOR PROTEIN P53; TYROSINE;

ATAXIA; ATAXIA TELANGIECTASIA;

EID: 0035905742 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1204111 Document Type: Article

Times cited : (67)

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