Pathways that suppress programmed DNA breaks from progressing to chromosomal breaks and translocations

DNA Repair

Volumn 5, Issue 9-10, 2006, Pages 1030-1041

  Franco, Sonia ^a,b   Alt, Frederick W ^a,b   Manis, John P ^a  

^a BOSTON CHILDREN S HOSPITAL   (United States)

^b CENTER FOR BLOOD RESEARCH   (United States)

Author keywords

Class switch recombination; DNA damage response; Lymphoma

Indexed keywords

DOUBLE STRANDED DNA; IMMUNOGLOBULIN HEAVY CHAIN; POLYDEOXYRIBONUCLEOTIDE SYNTHASE; PROTEIN P53;

APOPTOSIS; ARTICLE; B CELL LYMPHOMA; B LYMPHOCYTE; CELL CYCLE; CELL CYCLE G0 PHASE; CELL CYCLE G1 PHASE; CHROMOSOME ABERRATION; CHROMOSOME BREAKAGE; CHROMOSOME TRANSLOCATION; DNA REPAIR; DNA REPLICATION; DNA STRAND BREAKAGE; DOWN REGULATION; GENE LOCATION; GENE MUTATION; GENETIC RECOMBINATION; GENOMICS; GROWTH RETARDATION; HOMOLOGOUS RECOMBINATION; HUMAN; IMMUNITY; IONIZING RADIATION; LYMPHOCYTE; MONITORING; NONHUMAN; OXIDATIVE STRESS; PHENOTYPE; PHOSPHORYLATION; PRIORITY JOURNAL; PROTEIN DEFICIENCY; PROTEIN DEPHOSPHORYLATION; SEQUENCE HOMOLOGY; T CELL LYMPHOMA; T LYMPHOCYTE;

ANIMALS; CELL CYCLE PROTEINS; CHROMOSOME BREAKAGE; DNA DAMAGE; DNA REPAIR; DNA-BINDING PROTEINS; GENOMIC INSTABILITY; HISTONES; HOMEODOMAIN PROTEINS; HUMANS; LYMPHOCYTES; LYMPHOMA, B-CELL; PROTEIN-SERINE-THREONINE KINASES; TRANSLOCATION, GENETIC; TUMOR SUPPRESSOR PROTEIN P53; TUMOR SUPPRESSOR PROTEINS;

EID: 33748144349     PISSN: 15687864     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.dnarep.2006.05.024     Document Type: Article

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