H2AX may function as an anchor to hold broken chromosomal DNA ends in close proximity.

Cell cycle (Georgetown, Tex.)

Volumn 3, Issue 2, 2004, Pages 149-153

  Bassing, Craig H ^a   Alt, Frederick W ^a  

^a BOSTON CHILDREN S HOSPITAL   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ATM PROTEIN; CELL CYCLE PROTEIN; DNA BINDING PROTEIN; H2AFX PROTEIN, HUMAN; HISTONE; PROTEIN P53; PROTEIN SERINE THREONINE KINASE; TUMOR SUPPRESSOR PROTEIN;

ANIMAL; ATAXIA TELANGIECTASIA; BIOLOGICAL MODEL; CELL TRANSFORMATION; CHROMATIN; DNA DAMAGE; GENETICS; HUMAN; METABOLISM; MOUSE; MOUSE MUTANT; PHOSPHORYLATION; PHYSIOLOGY; REVIEW;

ANIMALS; ATAXIA TELANGIECTASIA; CELL CYCLE PROTEINS; CELL TRANSFORMATION, NEOPLASTIC; CHROMATIN; DNA DAMAGE; DNA-BINDING PROTEINS; HISTONES; HUMANS; MICE; MICE, KNOCKOUT; MODELS, BIOLOGICAL; PHOSPHORYLATION; PROTEIN-SERINE-THREONINE KINASES; TUMOR SUPPRESSOR PROTEIN P53; TUMOR SUPPRESSOR PROTEINS;

EID: 2442706035     PISSN: 15384101     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Review

References (47)