Rheumatoid arthritis onset in postmenopausal women: Does the ACPA seropositive subset result from genetic effects, estrogen deficiency, skewed profile of CD4+ T-cells, and their interactions?

Molecular and Cellular Endocrinology

Volumn 431, Issue , 2016, Pages 145-163

  Sapir Koren, Rony ^a   Livshits, Gregory ^a  

^a TEL AVIV UNIVERSITY   (Israel)

Author keywords

ACPA; Cytokines; Estrogens; Gene environment interactions; HLA DRB1 SE; Smoking

Indexed keywords

CYCLIC CITRULLINATED PEPTIDE ANTIBODY; ESTROGEN; ANTIBODY;

ANTIBODY DETECTION; ANTIBODY PRODUCTION; AUTOIMMUNITY; CD4+ T LYMPHOCYTE; CYTOKINE PRODUCTION; DISEASE ASSOCIATION; ESTROGEN BLOOD LEVEL; ESTROGEN DEFICIENCY; GENETIC POLYMORPHISM; GENETIC PREDISPOSITION; GENETIC RISK; GENOTYPE ENVIRONMENT INTERACTION; GENOTYPE PHENOTYPE CORRELATION; HUMAN; HUMORAL IMMUNITY; IMMUNOREACTIVITY; LYMPHOCYTE DIFFERENTIATION; LYMPHOCYTE FUNCTION; NONHUMAN; PERIODONTAL DISEASE; PORPHYROMONAS GINGIVALIS; POSTMENOPAUSE; PRIORITY JOURNAL; REGULATORY T LYMPHOCYTE; REVIEW; RHEUMATOID ARTHRITIS; SEX DIFFERENCE; SMOKING; T LYMPHOCYTE; TH1 CELL; TH17 CELL; FEMALE; IMMUNOLOGY;

ANTIBODIES; ARTHRITIS, RHEUMATOID; CD4-POSITIVE T-LYMPHOCYTES; ESTROGENS; FEMALE; HUMANS; POSTMENOPAUSE;

EID: 84973908636     PISSN: 03037207     EISSN: 18728057     Source Type: Journal    
DOI: 10.1016/j.mce.2016.05.009     Document Type: Review

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