An Immunochip-based interaction study of contrasting interaction effects with smoking in ACPA-positive versus ACPA-negative rheumatoid arthritis

Rheumatology (United Kingdom)

Volumn 55, Issue 1, 2016, Pages 149-155

  Jiang, Xia ^a   Källberg, Henrik ^a   Chen, Zuomei ^a   Ärlestig, Lisbeth ^b   Rantapää Dahlqvist, Solbritt ^b   Davila, Sonia ^c   Klareskog, Lars ^a   Padyukov, Leonid ^a   Alfredsson, Lars ^a,d  

^a KAROLINSKA INSTITUTE   (Sweden)

^b UMEÅ UNIVERSITY   (Sweden)

^c GENOME INSTITUTE OF SINGAPORE   (Singapore)

^d STOCKHOLM COUNTY COUNCIL   (Sweden)

Author keywords

biological interaction; HLA DRB1 shared epitope; Immunochip; Rheumatoid arthritis; smoking

Indexed keywords

CYCLIC CITRULLINATED PEPTIDE ANTIBODY; EPITOPE; MAJOR HISTOCOMPATIBILITY ANTIGEN CLASS 2;

ARTICLE; BTNL2 GENE; CHROMOSOME 6; CONTROLLED STUDY; GENE; GENE INTERACTION; GENE LOCUS; GENETIC ASSOCIATION; GENETIC MARKER; GENETIC VARIABILITY; GENOTYPE ENVIRONMENT INTERACTION; HLA DOB GENE; HLA DQA1 GENE; HLA DRA GENE; HLA DRB1 GENE; HLA DRB5 GENE; HLA SYSTEM; HUMAN; MAJOR CLINICAL STUDY; PRIORITY JOURNAL; RHEUMATOID ARTHRITIS; SINGLE NUCLEOTIDE POLYMORPHISM; SMOKING; SWEDEN; TAP2 GENE;

EID: 84960109796     PISSN: 14620324     EISSN: 14620332     Source Type: Journal    
DOI: 10.1093/rheumatology/kev285     Document Type: Article

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