The biological basis for cardiac repair after myocardial infarction

Circulation Research

Volumn 119, Issue 1, 2016, Pages 91-112

  Prabhu, Sumanth D ^a   Frangogiannis, Nikolaos G ^b  

^a University of Alabama at Birmingham   (United States)

^b ALBERT EINSTEIN COLLEGE OF MEDICINE   (United States)

Author keywords

chemokines; cytokines; fibrosis; immune cells; inflammation; myocardial infarction; myocytes, cardiac

Indexed keywords

ADVANCED GLYCATION END PRODUCT RECEPTOR; CHEMOKINE; CYTOKINE; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; NUCLEOTIDE BINDING OLIGOMERIZATION DOMAIN LIKE RECEPTOR; TOLL LIKE RECEPTOR;

CARDIAC REPAIR; CARDITIS; CELL PROLIFERATION; CELL TRANSDIFFERENTIATION; ENDOTHELIUM CELL; EXTRACELLULAR MATRIX; HEART FAILURE; HEART FIBROBLAST; HEART INFARCTION; HEART MUSCLE FIBROSIS; IMMUNE DYSREGULATION; INFLAMMATORY CELL; LYMPHOCYTE; MACROPHAGE; MAST CELL; MONOCYTE; MYOFIBROBLAST; NEUTROPHIL; PATHOGENESIS; PRIORITY JOURNAL; REVIEW; SIGNAL TRANSDUCTION; TISSUE REPAIR; ANIMAL; CARDIAC MUSCLE CELL; FIBROSIS; HUMAN; INFLAMMATION; METABOLISM; PATHOLOGY; PHYSIOLOGY; REGENERATION;

ANIMALS; CELL PROLIFERATION; FIBROSIS; HUMANS; INFLAMMATION; MYOCARDIAL INFARCTION; MYOCYTES, CARDIAC; REGENERATION;

EID: 84975818569     PISSN: 00097330     EISSN: 15244571     Source Type: Journal    
DOI: 10.1161/CIRCRESAHA.116.303577     Document Type: Review

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