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Volumn 107, Issue 3, 2010, Pages 418-428
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Smad3 signaling critically regulates fibroblast phenotype and function in healing myocardial infarction
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Author keywords
Fibroblasts; Fibrosis; Growth factors; Myocardial infarction; Transforming growth factor
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Indexed keywords
ALPHA SMOOTH MUSCLE ACTIN;
COLLAGEN;
CONNECTIVE TISSUE GROWTH FACTOR;
SCLEROPROTEIN;
SMAD3 PROTEIN;
TRANSFORMING GROWTH FACTOR BETA1;
SMAD3 PROTEIN, MOUSE;
TRANSACTIVATOR PROTEIN;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ARTICLE;
CELL FUNCTION;
CELL INFILTRATION;
CELL PROLIFERATION;
CONTROLLED STUDY;
FIBROBLAST;
HEART INFARCTION;
IN VITRO STUDY;
MOUSE;
MYOFIBROBLAST;
NONHUMAN;
PHENOTYPE;
PRIORITY JOURNAL;
PROTEIN EXPRESSION;
PROTEIN SYNTHESIS;
SIGNAL TRANSDUCTION;
UPREGULATION;
WILD TYPE;
ANIMAL;
BIOSYNTHESIS;
CELL DIFFERENTIATION;
CELL DIVISION;
CELL MOTION;
CYTOLOGY;
DRUG EFFECT;
GENETICS;
HEART CONTRACTION;
METABOLISM;
MOUSE MUTANT;
PATHOPHYSIOLOGY;
PHYSIOLOGY;
WOUND HEALING;
ANIMALS;
CELL DIFFERENTIATION;
CELL DIVISION;
CELL MOVEMENT;
EXTRACELLULAR MATRIX PROTEINS;
FIBROBLASTS;
MICE;
MICE, KNOCKOUT;
MYOCARDIAL CONTRACTION;
MYOCARDIAL INFARCTION;
PHENOTYPE;
SMAD3 PROTEIN;
TRANS-ACTIVATORS;
TRANSFORMING GROWTH FACTOR BETA1;
UP-REGULATION;
WOUND HEALING;
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EID: 77956627158
PISSN: 00097330
EISSN: 15244571
Source Type: Journal
DOI: 10.1161/CIRCRESAHA.109.216101 Document Type: Article |
Times cited : (320)
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References (5)
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