-
1
-
-
80155198133
-
The role of monocytes and inflammation in the pathophysiology of heart failure
-
Wrigley BJ Lip GY Shantsila E. The role of monocytes and inflammation in the pathophysiology of heart failure. Eur J Heart Fail 2011;13:1161-1171
-
(2011)
Eur J Heart Fail
, vol.13
, pp. 1161-1171
-
-
Wrigley, B.J.1
Lip, G.Y.2
Shantsila, E.3
-
2
-
-
84951924017
-
The cardiomyocyte as a source of cytokines in cardiac injury
-
Aoyagi T Matsui T. The cardiomyocyte as a source of cytokines in cardiac injury. J Cell Sci Ther 2011;S5:1-3
-
(2011)
J Cell Sci Ther
, vol.S5
, pp. 1-3
-
-
Aoyagi, T.1
Matsui, T.2
-
3
-
-
23944466382
-
Diastolic dysfunction in hypertensive hearts: Roles of perivascular inflammation and reactive myocardial fibrosis
-
Kai H Kuwahara F Tokuda K Imaizumi T. Diastolic dysfunction in hypertensive hearts: Roles of perivascular inflammation and reactive myocardial fibrosis. Hypertens Res 2005; 28:483-490
-
(2005)
Hypertens Res
, vol.28
, pp. 483-490
-
-
Kai, H.1
Kuwahara, F.2
Tokuda, K.3
Imaizumi, T.4
-
5
-
-
79955601433
-
The emerging role of innate immunity in the heart and vascular system: For whom the cell tolls
-
Mann DL. The emerging role of innate immunity in the heart and vascular system: For whom the cell tolls. Circ Res 2011;108:1133-1145
-
(2011)
Circ Res
, vol.108
, pp. 1133-1145
-
-
Mann, D.L.1
-
6
-
-
84869758819
-
Cardiokines: Recent progress in elucidating the cardiac secretome
-
Shimano M Ouchi NWalsh K. Cardiokines: Recent progress in elucidating the cardiac secretome. Circulation 2012;126:e327-e332
-
(2012)
Circulation
, vol.126
-
-
Shimano, M.1
Ouchi, N.2
Walsh, K.3
-
7
-
-
4544265332
-
Duality of innate stress responses in cardiac injury repair and remodeling
-
Wilson EM Diwan A Spinale FG Mann DL. Duality of innate stress responses in cardiac injury repair and remodeling. J Mol Cell Cardiol 2004;37:801-811
-
(2004)
J Mol Cell Cardiol
, vol.37
, pp. 801-811
-
-
Wilson, E.M.1
Diwan, A.2
Spinale, F.G.3
Mann, D.L.4
-
8
-
-
1242336786
-
Reduced myocardial ischemia-reperfusion injury in toll-like receptor 4-deficient mice
-
Oyama J Blais C Jr Liu X Pu M Kobzik L Kelly RA et al. Reduced myocardial ischemia-reperfusion injury in toll-like receptor 4-deficient mice. Circulation 2004;109: 784-789
-
(2004)
Circulation
, vol.109
, pp. 784-789
-
-
Oyama, J.1
Blais Jr., C.2
Liu, X.3
Pu, M.4
Kobzik, L.5
Kelly, R.A.6
-
9
-
-
41149165830
-
Toll-like receptor 4 mediates maladaptive left ventricular remodeling and impairs cardiac function after myocardial infarction
-
Timmers L Sluijter JP van Keulen JK Hoefer IE Nederhoff MG Goumans MJ et al. Toll-like receptor 4 mediates maladaptive left ventricular remodeling and impairs cardiac function after myocardial infarction. Circ Res 2008;102:257-264
-
(2008)
Circ Res
, vol.102
, pp. 257-264
-
-
Timmers, L.1
Sluijter, J.P.2
Van Keulen, J.K.3
Hoefer, I.E.4
Nederhoff, M.G.5
Goumans, M.J.6
-
10
-
-
79551502255
-
Peroxynitrite induces HMGB1 release by cardiac cells in vitro and HMGB1 upregulation in the infarcted myocardium in vivo
-
Loukili N Rosenblatt-Velin N Li J Clerc S Pacher P Feihl F et al. Peroxynitrite induces HMGB1 release by cardiac cells in vitro and HMGB1 upregulation in the infarcted myocardium in vivo. Cardiovasc Res 2011;89:586-594
-
(2011)
Cardiovasc Res
, vol.89
, pp. 586-594
-
-
Loukili, N.1
Rosenblatt-Velin, N.2
Li, J.3
Clerc, S.4
Pacher, P.5
Feihl, F.6
-
11
-
-
54449095004
-
Modulated inflammation by injection of high-mobility group box 1 recovers post-infarction chronically failing heart
-
Takahashi K Fukushima S Yamahara K YashiroK ShintaniYCoppen SR et al. Modulated inflammation by injection of high-mobility group box 1 recovers post-infarction chronically failing heart. Circulation 2008;118:S106-S114
-
(2008)
Circulation
, vol.118
-
-
Takahashi, K.1
Fukushima, S.2
Yamahara, K.3
Yashiro, K.4
Shintani, Y.5
Coppen, S.R.6
-
12
-
-
84860361435
-
Hmgb1 promotes recruitment of inflammatory cells to damaged tissues by forming a complex with cxcl12 and signaling via cxcr4
-
Schiraldi M Raucci A MunozLM Livoti E Celona B Venereau E et al.HMGB1 promotes recruitment of inflammatory cells to damaged tissues by forming a complex with CXCL12 and signaling via CXCR4. J Exp Med 2012;209:551-563
-
(2012)
J Exp Med
, vol.209
, pp. 551-563
-
-
Schiraldi, M.1
Raucci, A.2
Munoz, L.M.3
Livoti, E.4
Celona, B.5
Venereau, E.6
-
14
-
-
84875224962
-
Ca2+/calmodulindependent protein kinase II delta mediates myocardial ischemia/reperfusion injury through nuclear factor-kappaB
-
Ling H Gray CB Zambon AC Grimm M Gu Y Dalton N et al. Ca2+/calmodulindependent protein kinase II delta mediates myocardial ischemia/reperfusion injury through nuclear factor-kappaB. Circ Res 2013;112:935-944
-
(2013)
Circ Res
, vol.112
, pp. 935-944
-
-
Ling, H.1
Gray, C.B.2
Zambon, A.C.3
Grimm, M.4
Gu, Y.5
Dalton, N.6
-
15
-
-
79959527987
-
Calcineurin splicing variant calcineurin Abeta1 improves cardiac function after myocardial infarction without inducing hypertrophy
-
Felkin LE NaritaT Germack R ShintaniY Takahashi K Sarathchandra P et al. Calcineurin splicing variant calcineurin Abeta1 improves cardiac function after myocardial infarction without inducing hypertrophy. Circulation 2011;123:2838-2847
-
(2011)
Circulation
, vol.123
, pp. 2838-2847
-
-
Felkin, L.E.1
Narita, T.2
Germack, R.3
Shintani, Y.4
Takahashi, K.5
Sarathchandra, P.6
-
16
-
-
79955703474
-
GDF-15 is an inhibitor of leukocyte integrin activation required for survival after myocardial infarction in mice
-
Kempf T Zarbock A Widera C Butz S Stadtmann A Rossaint J et al. GDF-15 is an inhibitor of leukocyte integrin activation required for survival after myocardial infarction in mice. Nat Med 2011;17:581-588
-
(2011)
Nat Med
, vol.17
, pp. 581-588
-
-
Kempf, T.1
Zarbock, A.2
Widera, C.3
Butz, S.4
Stadtmann, A.5
Rossaint, J.6
-
18
-
-
0037067710
-
Contribution of macrophage migration inhibitory factor to extracellular signal-regulated kinase activation by oxidative stress in cardiomyocytes
-
Fukuzawa J Nishihira J Hasebe N Haneda T Osaki J Saito T et al. Contribution of macrophage migration inhibitory factor to extracellular signal-regulated kinase activation by oxidative stress in cardiomyocytes. J Biol Chem 2002;277:24889-24895
-
(2002)
J Biol Chem
, vol.277
, pp. 24889-24895
-
-
Fukuzawa, J.1
Nishihira, J.2
Hasebe, N.3
Haneda, T.4
Osaki, J.5
Saito, T.6
-
19
-
-
84883300963
-
Compartmentalized protective and detrimental effects of endogenous macrophage migrationinhibitory factor mediated by CXCR2 in a mouse model of myocardial ischemia/ reperfusion
-
Liehn EA Kanzler I Konschalla S Kroh A Simsekyilmaz S Sonmez TT et al. Compartmentalized protective and detrimental effects of endogenous macrophage migrationinhibitory factor mediated by CXCR2 in a mouse model of myocardial ischemia/ reperfusion. Arterioscler Thromb Vasc Biol 2013;33:2180-2186
-
(2013)
Arterioscler Thromb Vasc Biol
, vol.33
, pp. 2180-2186
-
-
Liehn, E.A.1
Kanzler, I.2
Konschalla, S.3
Kroh, A.4
Simsekyilmaz, S.5
Sonmez, T.T.6
-
20
-
-
65649138005
-
Endothelial nitric oxide synthase promotes bone marrowstromal cell migration to the ischemic myocardium via upregulation of stromal cell-derived factor-1alpha
-
Li N Lu X Zhao X Xiang FL Xenocostas A Karmazyn M et al. Endothelial nitric oxide synthase promotes bone marrowstromal cell migration to the ischemic myocardium via upregulation of stromal cell-derived factor-1alpha. Stem Cells 2009;27:961-970
-
(2009)
Stem Cells
, vol.27
, pp. 961-970
-
-
Li, N.1
Lu, X.2
Zhao, X.3
Xiang, F.L.4
Xenocostas, A.5
Karmazyn, M.6
-
22
-
-
84860705893
-
MitochondrialDNA that escapes from autophagy causes inflammation and heart failure
-
Oka T Hikoso S Yamaguchi O Taneike M Takeda T Tamai T et al. MitochondrialDNA that escapes from autophagy causes inflammation and heart failure. Nature 2012;485: 251-255
-
(2012)
Nature
, vol.485
, pp. 251-255
-
-
Oka, T.1
Hikoso, S.2
Yamaguchi, O.3
Taneike, M.4
Takeda, T.5
Tamai, T.6
-
24
-
-
77956867636
-
Intracellular or extracellular heat shock protein 70 differentially regulates cardiac remodelling in pressure overload mice
-
CaiWF ZhangXW Yan HM Ma YGWang XX Yan J et al. Intracellular or extracellular heat shock protein 70 differentially regulates cardiac remodelling in pressure overload mice. Cardiovasc Res 2010;88:140-149
-
(2010)
Cardiovasc Res
, vol.88
, pp. 140-149
-
-
Cai, W.F.1
Zhang, X.W.2
Yan, H.M.3
Ma, Y.G.4
Wang, X.X.5
Yan, J.6
-
25
-
-
77949315708
-
The alarmin cytokine high mobility group box 1 is produced by viable cardiomyocytes and mediates the lipopolysaccharide-induced myocardial dysfunction via a TLR4/phosphatidylinositol 3-kinase gamma pathway
-
Xu H Su Z Wu J Yang M Penninger JM Martin CM et al. The alarmin cytokine high mobility group box 1 is produced by viable cardiomyocytes and mediates the lipopolysaccharide-induced myocardial dysfunction via a TLR4/ phosphatidylinositol 3-kinase gamma pathway. J Immunol 2010;184:1492-1498
-
(2010)
J Immunol
, vol.184
, pp. 1492-1498
-
-
Xu, H.1
Su, Z.2
Wu, J.3
Yang, M.4
Penninger, J.M.5
Martin, C.M.6
-
26
-
-
77952290632
-
A new regulation of IL-6 production in adult cardiomyocytes by beta-Adrenergic and IL-1 beta receptors and induction of cellular hypertrophy by IL-6 trans-signalling
-
Szabo-Fresnais N Lefebvre F Germain A Fischmeister R Pomerance M. A new regulation of IL-6 production in adult cardiomyocytes by beta-Adrenergic and IL-1 beta receptors and induction of cellular hypertrophy by IL-6 trans-signalling. Cell Signal 2010;22: 1143-1152
-
(2010)
Cell Signal
, vol.22
, pp. 1143-1152
-
-
Szabo-Fresnais, N.1
Lefebvre, F.2
Germain, A.3
Fischmeister, R.4
Pomerance, M.5
-
27
-
-
1642305705
-
Inhibition of p38 MAPK decreases myocardial TNF-Alpha expression and improves myocardial function and survival in endotoxemia
-
Peng T Lu X Lei M Moe GW Feng Q. Inhibition of p38 MAPK decreases myocardial TNF-Alpha expression and improves myocardial function and survival in endotoxemia. Cardiovasc Res 2003;59:893-900
-
(2003)
Cardiovasc Res
, vol.59
, pp. 893-900
-
-
Peng, T.1
Lu, X.2
Lei, M.3
Moe, G.W.4
Feng, Q.5
-
28
-
-
60149085989
-
JNK1/c-fos inhibits cardiomyocyte TNF-Alpha expression via a negative crosstalk with ERK and p38 MAPK in endotoxaemia
-
PengT ZhangT LuX Feng Q. JNK1/c-fos inhibits cardiomyocyte TNF-Alpha expression via a negative crosstalk with ERK and p38 MAPK in endotoxaemia. Cardiovasc Res 2009; 81:733-741
-
(2009)
Cardiovasc Res
, vol.81
, pp. 733-741
-
-
Peng, T.1
Zhang, T.2
Lu, X.3
Feng, Q.4
-
29
-
-
27144512843
-
Inhibitor-kappaB kinase-beta regulates LPS-induced TNF-Alpha production in cardiac myocytes through modulation of NF-kappaB p65 subunit phosphorylation
-
Hall G Singh IS Hester L Hasday JD Rogers TB. Inhibitor-kappaB kinase-beta regulates LPS-induced TNF-Alpha production in cardiac myocytes through modulation of NF-kappaB p65 subunit phosphorylation. Am J Physiol Heart Circ Physiol 2005;289: H2103-H2111
-
(2005)
Am J Physiol Heart Circ Physiol
, vol.289
-
-
Hall, G.1
Singh, I.S.2
Hester, L.3
Hasday, J.D.4
Rogers, T.B.5
-
30
-
-
34147098000
-
Tumor necrosis factor-Alpha mediates cardiac remodeling and ventricular dysfunction after pressure overload state
-
Sun M Chen M DawoodF Zurawska U Li JY ParkerTet al. Tumor necrosis factor-Alpha mediates cardiac remodeling and ventricular dysfunction after pressure overload state. Circulation 2007;115:1398-1407
-
(2007)
Circulation
, vol.115
, pp. 1398-1407
-
-
Sun, M.1
Chen, M.2
Dawood, F.3
Zurawska, U.4
Li, J.Y.5
Parker, T.6
-
31
-
-
0036198371
-
Tumor necrosis factoralpha and myocardial remodeling in progression of heart failure: A current perspective
-
Bradham WS Bozkurt B Gunasinghe H Mann D Spinale FG. Tumor necrosis factoralpha and myocardial remodeling in progression of heart failure: A current perspective. Cardiovasc Res 2002;53:822-830
-
(2002)
Cardiovasc Res
, vol.53
, pp. 822-830
-
-
Bradham, W.S.1
Bozkurt, B.2
Gunasinghe, H.3
Mann, D.4
Spinale, F.G.5
-
32
-
-
80855143703
-
The development of myocardial fibrosis in transgenic mice with targeted overexpression of tumor necrosis factor requires mast cell-fibroblast interactions
-
ZhangW Chancey AL Tzeng HP Zhou Z Lavine KJ Gao F et al. The development of myocardial fibrosis in transgenic mice with targeted overexpression of tumor necrosis factor requires mast cell-fibroblast interactions. Circulation 2011;124:2106-2116
-
(2011)
Circulation
, vol.124
, pp. 2106-2116
-
-
Zhang, W.1
Chancey, A.L.2
Tzeng, H.P.3
Zhou, Z.4
Lavine, K.J.5
Gao, F.6
-
34
-
-
84859038834
-
Receptor activator of nuclear factorkappaB ligand is a novel inducer of myocardial inflammation
-
Ock S Ahn J Lee SH Park H Son JW Oh JG et al. Receptor activator of nuclear factorkappaB ligand is a novel inducer of myocardial inflammation. Cardiovasc Res 2012;94: 105-114
-
(2012)
Cardiovasc Res
, vol.94
, pp. 105-114
-
-
Ock, S.1
Ahn, J.2
Lee, S.H.3
Park, H.4
Son, J.W.5
Oh, J.G.6
-
35
-
-
84864277489
-
Interleukin-10 treatment attenuates pressure overload-induced hypertrophic remodeling and improves heart function via signal transducers and activators of transcription 3-dependent inhibition of nuclear factor-kappaB
-
Verma SK Krishnamurthy P Barefield D Singh N Gupta R Lambers E et al. Interleukin-10 treatment attenuates pressure overload-induced hypertrophic remodeling and improves heart function via signal transducers and activators of transcription 3-dependent inhibition of nuclear factor-kappaB. Circulation 2012;126:418-429
-
(2012)
Circulation
, vol.126
, pp. 418-429
-
-
Verma, S.K.1
Krishnamurthy, P.2
Barefield, D.3
Singh, N.4
Gupta, R.5
Lambers, E.6
-
36
-
-
77956176871
-
EMMPRIN activates multiple transcription factors in cardiomyocytes and induces interleukin-18 expression via Rac1-dependent PI3K/Akt/IKK/NF-kappaB andMKK7/ JNK/AP-1 signaling
-
Venkatesan B Valente AJ Prabhu SD Shanmugam P Delafontaine P Chandrasekar B. EMMPRIN activates multiple transcription factors in cardiomyocytes and induces interleukin-18 expression via Rac1-dependent PI3K/Akt/IKK/NF-kappaB andMKK7/ JNK/AP-1 signaling. J Mol Cell Cardiol 2010;49:655-663
-
(2010)
J Mol Cell Cardiol
, vol.49
, pp. 655-663
-
-
Venkatesan, B.1
Valente, A.J.2
Prabhu, S.D.3
Shanmugam, P.4
Delafontaine, P.5
Chandrasekar, B.6
-
37
-
-
39449095307
-
Extracellular matrix metalloproteinase inducer (CD147) is a novel receptor on platelets activates platelets and augments nuclear factor kappaB-dependent inflammation in monocytes
-
Schmidt R Bultmann A Fischel S Gillitzer A Cullen PWalchAet al. Extracellular matrix metalloproteinase inducer (CD147) is a novel receptor on platelets activates platelets and augments nuclear factor kappaB-dependent inflammation in monocytes. Circ Res 2008;102:302-309
-
(2008)
Circ Res
, vol.102
, pp. 302-309
-
-
Schmidt, R.1
Bultmann, A.2
Fischel, S.3
Gillitzer, A.4
Cullen, P.5
Walch, A.6
-
38
-
-
67749099899
-
Lack of IL-6 during coxsackievirus infection heightens the early immune response resulting in increased severity of chronic autoimmune myocarditis
-
Poffenberger MC Straka N El Warry N Fang D Shanina I Horwitz MS. Lack of IL-6 during coxsackievirus infection heightens the early immune response resulting in increased severity of chronic autoimmune myocarditis. PLoS ONE 2009;4:e6207
-
(2009)
PLoS ONE
, vol.4
-
-
Poffenberger, M.C.1
Straka, N.2
El Warry, N.3
Fang, D.4
Shanina, I.5
Horwitz, M.S.6
-
39
-
-
7644244579
-
Coxsackievirus group B type 3 infection upregulates expression of monocyte chemoattractant protein 1 in cardiac myocytes which leads to enhanced migration of mononuclear cells in viral myocarditis
-
Shen Y XuW Chu YWWang Y Liu QS Xiong SD. Coxsackievirus group B type 3 infection upregulates expression of monocyte chemoattractant protein 1 in cardiac myocytes which leads to enhanced migration of mononuclear cells in viral myocarditis. J Virol 2004;78:12548-12556
-
(2004)
J Virol
, vol.78
, pp. 12548-12556
-
-
Shen, Y.1
Xu, W.2
Chu, Y.W.3
Wang, Y.4
Liu, Q.S.5
Xiong, S.D.6
-
40
-
-
63649114185
-
CXCL10 inhibits viral replication through recruitment of natural killer cells in coxsackievirus B3-induced myocarditis
-
Yuan J Liu Z Lim T Zhang H He J Walker E et al. CXCL10 inhibits viral replication through recruitment of natural killer cells in coxsackievirus B3-induced myocarditis. Circ Res 2009;104:628-638
-
(2009)
Circ Res
, vol.104
, pp. 628-638
-
-
Yuan, J.1
Liu, Z.2
Lim, T.3
Zhang, H.4
He, J.5
Walker, E.6
-
42
-
-
79953678927
-
The coxsackie-Adenovirus receptor induces an inflammatory cardiomyopathy independent of viral infection
-
Yuen S Smith J Caruso L Balan M Opavsky MA. The coxsackie-Adenovirus receptor induces an inflammatory cardiomyopathy independent of viral infection. J Mol Cell Cardiol 2011;50:826-840
-
(2011)
J Mol Cell Cardiol
, vol.50
, pp. 826-840
-
-
Yuen, S.1
Smith, J.2
Caruso, L.3
Balan, M.4
Opavsky, M.A.5
-
43
-
-
77952669050
-
Cardiomyocyte-Targeted overexpression of the coxsackie-Adenovirus receptor causes a cardiomyopathy in association with beta-catenin signaling
-
Caruso L Yuen S Smith J Husain M Opavsky MA. Cardiomyocyte-Targeted overexpression of the coxsackie-Adenovirus receptor causes a cardiomyopathy in association with beta-catenin signaling. J Mol Cell Cardiol 2010;48:1194-1205
-
(2010)
J Mol Cell Cardiol
, vol.48
, pp. 1194-1205
-
-
Caruso, L.1
Yuen, S.2
Smith, J.3
Husain, M.4
Opavsky, M.A.5
-
45
-
-
70149084792
-
HMGB1 loves company
-
Bianchi ME. HMGB1 loves company. J Leukoc Biol 2009;86:573-576
-
(2009)
J Leukoc Biol
, vol.86
, pp. 573-576
-
-
Bianchi, M.E.1
-
49
-
-
77954762928
-
Continuous glycoprotein-130-mediated signal transducer and activator of transcription-3 activation promotes inflammation left ventricular rupture and adverse outcome in subacute myocardial infarction
-
Hilfiker-Kleiner D Shukla P Klein G Schaefer A Stapel B Hoch M et al. Continuous glycoprotein-130-mediated signal transducer and activator of transcription-3 activation promotes inflammation left ventricular rupture and adverse outcome in subacute myocardial infarction. Circulation 2010;122:145-155
-
(2010)
Circulation
, vol.122
, pp. 145-155
-
-
Hilfiker-Kleiner, D.1
Shukla, P.2
Klein, G.3
Schaefer, A.4
Stapel, B.5
Hoch, M.6
-
50
-
-
3342889421
-
Signal transducer and activator of transcription 3 is required for myocardial capillary growth control of interstitial matrix deposition and heart protection from ischemic injury
-
Hilfiker-Kleiner D Hilfiker A Fuchs M Kaminski K Schaefer A Schieffer B et al. Signal transducer and activator of transcription 3 is required for myocardial capillary growth control of interstitial matrix deposition and heart protection from ischemic injury. Circ Res 2004;95:187-195
-
(2004)
Circ Res
, vol.95
, pp. 187-195
-
-
Hilfiker-Kleiner, D.1
Hilfiker, A.2
Fuchs, M.3
Kaminski, K.4
Schaefer, A.5
Schieffer, B.6
-
51
-
-
77952738714
-
Increased production of CXCL16 in experimental and clinical heart failure: A possible role in extracellular matrix remodeling
-
Dahl CP Husberg C Gullestad LWaehre A Damas JK Vinge LE et al. Increased production of CXCL16 in experimental and clinical heart failure: A possible role in extracellular matrix remodeling. Circ Heart Fail 2009;2:624-632
-
(2009)
Circ Heart Fail
, vol.2
, pp. 624-632
-
-
Dahl, C.P.1
Husberg, C.2
Gullestad, L.3
Waehre, A.4
Damas, J.K.5
Vinge, L.E.6
-
52
-
-
33646076691
-
The transforming growth factor-beta superfamily member growth-differentiation factor-15 protects the heart from ischemia/reperfusion injury
-
Kempf T Eden M Strelau J Naguib M Willenbockel C Tongers J et al. The transforming growth factor-beta superfamily member growth-differentiation factor-15 protects the heart from ischemia/reperfusion injury. Circ Res 2006;98:351-360
-
(2006)
Circ Res
, vol.98
, pp. 351-360
-
-
Kempf, T.1
Eden, M.2
Strelau, J.3
Naguib, M.4
Willenbockel, C.5
Tongers, J.6
-
53
-
-
84873868258
-
Signaling effectors underlying pathologic growth and remodeling of the heart
-
van Berlo JH Maillet M Molkentin JD. Signaling effectors underlying pathologic growth and remodeling of the heart. J Clin Invest 2013;123:37-45
-
(2013)
J Clin Invest
, vol.123
, pp. 37-45
-
-
Van Berlo, J.H.1
Maillet, M.2
Molkentin, J.D.3
-
55
-
-
84869396139
-
Priming with synthetic oligonucleotides attenuates pressure overload-induced inflammation and cardiac hypertrophy in mice
-
Velten M Duerr GD Pessies T Schild J Lohner R Mersmann J et al. Priming with synthetic oligonucleotides attenuates pressure overload-induced inflammation and cardiac hypertrophy in mice. Cardiovasc Res 2012;96:422-432
-
(2012)
Cardiovasc Res
, vol.96
, pp. 422-432
-
-
Velten, M.1
Duerr, G.D.2
Pessies, T.3
Schild, J.4
Lohner, R.5
Mersmann, J.6
-
56
-
-
84877934501
-
Extracellular HSP60 induces inflammation through activating and up-regulating TLRs in cardiomyocytes
-
Tian J Guo X Liu XM Liu L Weng QF Dong SJ et al. Extracellular HSP60 induces inflammation through activating and up-regulating TLRs in cardiomyocytes. Cardiovasc Res 2013;98:391-401
-
(2013)
Cardiovasc Res
, vol.98
, pp. 391-401
-
-
Tian, J.1
Guo, X.2
Liu, X.M.3
Liu, L.4
Weng, Q.F.5
Dong, S.J.6
-
58
-
-
84867881959
-
Phosphoinositide 3-kinase gamma protects against catecholamine-induced ventricular arrhythmia through protein kinase A-mediated regulation of distinct phosphodiesterases
-
Ghigo A Perino A Mehel H ZahradnikovaAJr Morello F Leroy J et al. Phosphoinositide 3-kinase gamma protects against catecholamine-induced ventricular arrhythmia through protein kinase A-mediated regulation of distinct phosphodiesterases. Circulation 2012; 126:2073-2083
-
(2012)
Circulation
, vol.126
, pp. 2073-2083
-
-
Ghigo, A.1
Perino, A.2
Mehel, H.3
Zahradnikova Jr., A.4
Morello, F.5
Leroy, J.6
-
60
-
-
79751524816
-
Distinct effects of leukocyte and cardiac phosphoinositide 3-kinase gamma activity in pressure overload-induced cardiac failure
-
Damilano F Franco I Perrino C Schaefer K Azzolino O Carnevale D et al. Distinct effects of leukocyte and cardiac phosphoinositide 3-kinase gamma activity in pressure overload-induced cardiac failure. Circulation 2011;123:391-399
-
(2011)
Circulation
, vol.123
, pp. 391-399
-
-
Damilano, F.1
Franco, I.2
Perrino, C.3
Schaefer, K.4
Azzolino, O.5
Carnevale, D.6
-
61
-
-
67449161762
-
Angiotensin II downregulates the fatty acid oxidation pathway in adult rat cardiomyocytes via release of tumour necrosis factoralpha
-
Pellieux C Montessuit C Papageorgiou I Lerch R. Angiotensin II downregulates the fatty acid oxidation pathway in adult rat cardiomyocytes via release of tumour necrosis factoralpha. Cardiovasc Res 2009;82:341-350
-
(2009)
Cardiovasc Res
, vol.82
, pp. 341-350
-
-
Pellieux, C.1
Montessuit, C.2
Papageorgiou, I.3
Lerch, R.4
-
62
-
-
84873044964
-
Cross-Talk between inflammation and angiotensin II: Studies based on Direct transfection of cardiomyocytes withAT1R and AT2R cDNA
-
Wang X Khaidakov M Ding Z Mitra S Lu J Liu S et al. Cross-Talk between inflammation and angiotensin II: Studies based on Direct transfection of cardiomyocytes withAT1R and AT2R cDNA. Exp Biol Med (Maywood) 2012;237:1394-1401
-
(2012)
Exp Biol Med (Maywood
, vol.237
, pp. 1394-1401
-
-
Wang, X.1
Khaidakov, M.2
Ding, Z.3
Mitra, S.4
Lu, J.5
Liu, S.6
-
63
-
-
41149108014
-
Disruption of phospholipaseCgamma1 signalling attenuates cardiac tumor necrosis factor-Alpha expression and improves myocardial function during endotoxemia
-
PengT Shen E Fan J ZhangY Arnold JM Feng Q. Disruption of phospholipaseCgamma1 signalling attenuates cardiac tumor necrosis factor-Alpha expression and improves myocardial function During endotoxemia. Cardiovasc Res 2008;78:90-97
-
(2008)
Cardiovasc Res
, vol.78
, pp. 90-97
-
-
Peng, T.1
Shen, E.2
Fan, J.3
Zhang, Y.4
Arnold, J.M.5
Feng, Q.6
-
64
-
-
77951226550
-
Histone deacetylase-3 activation promotes tumor necrosis factor-Alpha (TNF-Alpha) expression in cardiomyocytes during lipopolysaccharide stimulation
-
Zhu H Shan L Schiller PW Mai A Peng T. Histone deacetylase-3 activation promotes tumor necrosis factor-Alpha (TNF-Alpha) expression in cardiomyocytes during lipopolysaccharide stimulation. J Biol Chem 2010;285:9429-9436
-
(2010)
J Biol Chem
, vol.285
, pp. 9429-9436
-
-
Zhu, H.1
Shan, L.2
Schiller, P.W.3
Mai, A.4
Peng, T.5
-
65
-
-
34250736360
-
Role of neuronal nitric oxide synthase in lipopolysaccharide-induced tumor necrosis factor-Alpha expression in neonatal mouse cardiomyocytes
-
Geoghegan-Morphet N Burger D Lu X SathishV PengT SimsSMet al. Role of neuronal nitric oxide synthase in lipopolysaccharide-induced tumor necrosis factor-Alpha expression in neonatal mouse cardiomyocytes. Cardiovasc Res 2007;75:408-416
-
(2007)
Cardiovasc Res
, vol.75
, pp. 408-416
-
-
Geoghegan-Morphet, N.1
Burger, D.2
Lu, X.3
Sathish, V.4
Peng, T.5
Sims, S.M.6
-
66
-
-
84863254909
-
Mitogen-Activated protein kinase phosphatase-1 inhibits myocardial TNF-Alpha expression and improves cardiac function during endotoxemia
-
Zhang T Lu X Arnold P Liu Y Baliga R Huang H et al. Mitogen-Activated protein kinase phosphatase-1 inhibits myocardial TNF-Alpha expression and improves cardiac function During endotoxemia. Cardiovasc Res 2012;93:471-479
-
(2012)
Cardiovasc Res
, vol.93
, pp. 471-479
-
-
Zhang, T.1
Lu, X.2
Arnold, P.3
Liu, Y.4
Baliga, R.5
Huang, H.6
-
67
-
-
46449107402
-
Glycogen synthase kinase-3beta suppresses tumor necrosis factoralpha expression in cardiomyocytes during lipopolysaccharide stimulation
-
Shen E Fan J Peng T. Glycogen synthase kinase-3beta suppresses tumor necrosis factoralpha expression in cardiomyocytes during lipopolysaccharide stimulation. J Cell Biochem 2008;104:329-338
-
(2008)
J Cell Biochem
, vol.104
, pp. 329-338
-
-
Shen, E.1
Fan, J.2
Peng, T.3
-
68
-
-
77952672109
-
Cardiomyocyte Toll-like receptor 4 is involved in heart dysfunction following septic shock or myocardial ischemia
-
Fallach R Shainberg A Avlas O Fainblut M Chepurko Y Porat E et al. Cardiomyocyte Toll-like receptor 4 is involved in heart dysfunction following septic shock or myocardial ischemia. J Mol Cell Cardiol 2010;48:1236-1244
-
(2010)
J Mol Cell Cardiol
, vol.48
, pp. 1236-1244
-
-
Fallach, R.1
Shainberg, A.2
Avlas, O.3
Fainblut, M.4
Chepurko, Y.5
Porat, E.6
-
69
-
-
0025286817
-
Elevated circulating levels of tumor necrosis factor in severe chronic heart failure
-
Levine B Kalman J Mayer L Fillit HM Packer M. Elevated circulating levels of tumor necrosis factor in severe chronic heart failure. N Engl J Med 1990;323:236-241
-
(1990)
N Engl J Med
, vol.323
, pp. 236-241
-
-
Levine, B.1
Kalman, J.2
Mayer, L.3
Fillit, H.M.4
Packer, M.5
-
70
-
-
11144355760
-
Targeted anticytokine therapy in patients with chronic heart failure: Results of the Randomized EtanerceptWorldwide Evaluation (RENEWAL
-
Mann DL McMurray JJ Packer M Swedberg K Borer JS Colucci WS et al. Targeted anticytokine therapy in patients with chronic heart failure: Results of the Randomized EtanerceptWorldwide Evaluation (RENEWAL). Circulation 2004;109:1594-1602
-
(2004)
Circulation
, vol.109
, pp. 1594-1602
-
-
Mann, D.L.1
McMurray, J.J.2
Packer, M.3
Swedberg, K.4
Borer, J.S.5
Colucci, W.S.6
-
71
-
-
0038755661
-
Randomized double-blind placebo-controlled pilot trial of infliximab a chimeric monoclonal antibody to tumor necrosis factor-Alpha in patients with moderate-To-severe heart failure: Results of the anti-Tnf therapy against congestive heart failure (attach) trial
-
Chung ES Packer M Lo KH Fasanmade AA Willerson JT. Randomized double-blind placebo-controlled pilot trial of infliximab a chimeric monoclonal antibody to tumor necrosis factor-Alpha in patients with moderate-To-severe heart failure: Results of the anti-TNF Therapy Against Congestive Heart Failure (ATTACH) trial. Circulation 2003; 107:3133-3140
-
(2003)
Circulation
, vol.107
, pp. 3133-3140
-
-
Chung, E.S.1
Packer, M.2
Lo, K.H.3
Fasanmade, A.A.4
Willerson, J.T.5
-
72
-
-
0034625134
-
Endogenous tumor necrosis factor protects the adult cardiac myocyte against ischemic-induced apoptosis in a murine model of acute myocardial infarction
-
Kurrelmeyer KM Michael LH Baumgarten G Taffet GE Peschon JJ Sivasubramanian N et al. Endogenous tumor necrosis factor protects the adult cardiac myocyte against ischemic-induced apoptosis in a murine model of acute myocardial infarction. Proc Natl Acad Sci USA 2000;97:5456-5461
-
(2000)
Proc Natl Acad Sci USA
, vol.97
, pp. 5456-5461
-
-
Kurrelmeyer, K.M.1
Michael, L.H.2
Baumgarten, G.3
Taffet, G.E.4
Peschon, J.J.5
Sivasubramanian, N.6
-
73
-
-
37549023499
-
Emmprin mediates beta-Adrenergic receptor-stimulated matrix metalloproteinase activity in cardiac myocytes
-
Siwik DA Kuster GM Brahmbhatt JV Zaidi Z Malik J Ooi H et al. EMMPRIN mediates beta-Adrenergic receptor-stimulated matrix metalloproteinase activity in cardiac myocytes. J Mol Cell Cardiol 2008;44:210-217
-
(2008)
J Mol Cell Cardiol
, vol.44
, pp. 210-217
-
-
Siwik, D.A.1
Kuster, G.M.2
Brahmbhatt, J.V.3
Zaidi, Z.4
Malik, J.5
Ooi, H.6
-
74
-
-
33751231104
-
Interplay of TNF-Alpha and IL-10 in regulating oxidative stress in isolated adult cardiac myocytes
-
Kaur K Sharma AK Dhingra S Singal PK. Interplay of TNF-Alpha and IL-10 in regulating oxidative stress in isolated adult cardiac myocytes. J Mol Cell Cardiol 2006;41:1023-1030
-
(2006)
J Mol Cell Cardiol
, vol.41
, pp. 1023-1030
-
-
Kaur, K.1
Sharma, A.K.2
Dhingra, S.3
Singal, P.K.4
-
75
-
-
84855371090
-
Regulation of the inflammatory response in cardiac repair
-
Frangogiannis NG. Regulation of the inflammatory response in cardiac repair. Circ Res 2012;110:159-173
-
(2012)
Circ Res
, vol.110
, pp. 159-173
-
-
Frangogiannis, N.G.1
-
77
-
-
64349106997
-
Myocarditis
-
Cooper LT Jr. Myocarditis. N Engl J Med 2009;360:1526-1538
-
(2009)
N Engl J Med
, vol.360
, pp. 1526-1538
-
-
Cooper Jr., L.T.1
-
78
-
-
0025191285
-
Induction of major histocompatibility complex antigens within the myocardium of patients with active myocarditis: A nonhistologic marker of myocarditis
-
Herskowitz A Ahmed-Ansari A Neumann DA Beschorner WE Rose NR Soule LM et al. Induction of major histocompatibility complex antigens within the myocardium of patients with active myocarditis: A nonhistologic marker of myocarditis. J Am Coll Cardiol 1990;15:624-632
-
(1990)
J Am Coll Cardiol
, vol.15
, pp. 624-632
-
-
Herskowitz, A.1
Ahmed-Ansari, A.2
Neumann, D.A.3
Beschorner, W.E.4
Rose, N.R.5
Soule, L.M.6
-
80
-
-
0031159090
-
Cardiac-specific overexpression of tumor necrosis factor-Alpha causes lethal myocarditis in transgenic mice
-
Kubota T McTiernan CF Frye CS Demetris AJ Feldman AM. Cardiac-specific overexpression of tumor necrosis factor-Alpha causes lethal myocarditis in transgenic mice. J Card Fail 1997;3:117-124
-
(1997)
J Card Fail
, vol.3
, pp. 117-124
-
-
Kubota, T.1
McTiernan, C.F.2
Frye, C.S.3
Demetris, A.J.4
Feldman, A.M.5
-
82
-
-
83455230796
-
Chronic inflammatory cardiomyopathy of interferon gamma-overexpressing transgenic mice is mediated by tumor necrosis factor-Alpha
-
Torzewski M Wenzel P Kleinert H Becker C El-Masri J Wiese E et al. Chronic inflammatory cardiomyopathy of interferon gamma-overexpressing transgenic mice is mediated by tumor necrosis factor-Alpha. Am J Pathol 2012;180:73-81
-
(2012)
Am J Pathol
, vol.180
, pp. 73-81
-
-
Torzewski, M.1
Wenzel, P.2
Kleinert, H.3
Becker, C.4
El-Masri, J.5
Wiese, E.6
-
83
-
-
0027250114
-
Role of IL-1 and tumor necrosis factor in coxsackie virus-induced autoimmune myocarditis
-
Lane JR Neumann DA Lafond-Walker A Herskowitz A Rose NR. Role of IL-1 and tumor necrosis factor in coxsackie virus-induced autoimmune myocarditis. J Immunol 1993;151:1682-1690
-
(1993)
J Immunol
, vol.151
, pp. 1682-1690
-
-
Lane, J.R.1
Neumann, D.A.2
Lafond-Walker, A.3
Herskowitz, A.4
Rose, N.R.5
-
84
-
-
70349257967
-
Differential macrophage polarization in male and female BALB/c mice infected with coxsackievirus B3 defines susceptibility to viral myocarditis
-
Li K XuW Guo Q Jiang ZWang P Yue Y et al. Differential macrophage polarization in male and female BALB/c mice infected with coxsackievirus B3 defines susceptibility to viral myocarditis. Circ Res 2009;105:353-364
-
(2009)
Circ Res
, vol.105
, pp. 353-364
-
-
Li, K.1
Xu, W.2
Guo, Q.3
Jiang, Z.4
Wang, P.5
Yue, Y.6
-
85
-
-
38749120919
-
Sustained nitric oxide synthesis contributes to immunopathology in ongoing myocarditis attributable to interleukin-10 disorders
-
Szalay G Sauter M Hald J Weinzierl A Kandolf R Klingel K. Sustained nitric oxide synthesis contributes to immunopathology in ongoing myocarditis attributable to interleukin-10 disorders. Am J Pathol 2006;169:2085-2093
-
(2006)
Am J Pathol
, vol.169
, pp. 2085-2093
-
-
Szalay, G.1
Sauter, M.2
Hald, J.3
Weinzierl, A.4
Kandolf, R.5
Klingel, K.6
-
87
-
-
0034849946
-
Overexpression of interleukin-6 aggravates viral myocarditis: Impaired increase in tumor necrosis factoralpha
-
Tanaka T Kanda TMcManus BM Kanai H Akiyama H Sekiguchi K et al. Overexpression of interleukin-6 aggravates viral myocarditis: Impaired increase in tumor necrosis factoralpha. J Mol Cell Cardiol 2001;33:1627-1635
-
(2001)
J Mol Cell Cardiol
, vol.33
, pp. 1627-1635
-
-
Tanaka, T.1
Kanda, T.2
McManus, B.M.3
Kanai, H.4
Akiyama, H.5
Sekiguchi, K.6
-
88
-
-
0034854396
-
Interleukin-6 and viral myocarditis: The Yin-Yang of cardiac innate immune responses
-
Mann DL. Interleukin-6 and viral myocarditis: The Yin-Yang of cardiac innate immune responses. J Mol Cell Cardiol 2001;33:1551-1553
-
(2001)
J Mol Cell Cardiol
, vol.33
, pp. 1551-1553
-
-
Mann, D.L.1
-
90
-
-
77954781995
-
Antibody against interleukin-6 receptor attenuates left ventricular remodelling after myocardial infarction in mice
-
Kobara M Noda K Kitamura M Okamoto A Shiraishi T Toba H et al. Antibody against interleukin-6 receptor attenuates left ventricular remodelling after myocardial infarction in mice. Cardiovasc Res 2010;87:424-430
-
(2010)
Cardiovasc Res
, vol.87
, pp. 424-430
-
-
Kobara, M.1
Noda, K.2
Kitamura, M.3
Okamoto, A.4
Shiraishi, T.5
Toba, H.6
-
91
-
-
84875812252
-
STAT3 activity is necessary and sufficient for the development of immune-mediated myocarditis in mice and promotes progression to dilated cardiomyopathy
-
Camporeale A Marino F Papageorgiou A Carai P Fornero S Fletcher S et al. STAT3 activity is necessary and sufficient for the development of immune-mediated myocarditis in mice and promotes progression to dilated cardiomyopathy. EMBO Mol Med 2013;5: 572-590
-
(2013)
EMBO Mol Med
, vol.5
, pp. 572-590
-
-
Camporeale, A.1
Marino, F.2
Papageorgiou, A.3
Carai, P.4
Fornero, S.5
Fletcher, S.6
-
92
-
-
65249125949
-
Osteopontin: A fibrosis-related marker molecule in cardiac remodeling of enterovirus myocarditis in the susceptible host
-
Szalay G Sauter M Haberland M Zuegel U Steinmeyer A KandolfRet al. Osteopontin: A fibrosis-related marker molecule in cardiac remodeling of enterovirus myocarditis in the susceptible host. Circ Res 2009;104:851-859
-
(2009)
Circ Res
, vol.104
, pp. 851-859
-
-
Szalay, G.1
Sauter, M.2
Haberland, M.3
Zuegel, U.4
Steinmeyer, A.5
Kandolf, R.6
-
93
-
-
41749107345
-
Molecular biology and pathogenesis of viral myocarditis
-
Esfandiarei M McManus BM. Molecular biology and pathogenesis of viral myocarditis. Annu Rev Pathol 2008;3:127-155
-
(2008)
Annu Rev Pathol
, vol.3
, pp. 127-155
-
-
Esfandiarei, M.1
McManus, B.M.2
-
94
-
-
63249122666
-
Cardiac deletion of the coxsackievirus-Adenovirus receptor abolishes coxsackievirus B3 infection and prevents myocarditis in vivo
-
Shi Y Chen C Lisewski U Wrackmeyer U Radke M Westermann D et al. Cardiac deletion of the coxsackievirus-Adenovirus receptor abolishes coxsackievirus B3 infection and prevents myocarditis in vivo. J Am Coll Cardiol 2009;53: 1219-1226
-
(2009)
J Am Coll Cardiol
, vol.53
, pp. 1219-1226
-
-
Shi, Y.1
Chen, C.2
Lisewski, U.3
Wrackmeyer, U.4
Radke, M.5
Westermann, D.6
-
95
-
-
40149095736
-
Activation of STAT1 transcription factor precedes up-regulation of coxsackievirus-Adenovirus receptor during viral myocarditis
-
Ruppert V Meyer T Pankuweit S Jonsdottir T Maisch B. Activation of STAT1 transcription factor precedes up-regulation of coxsackievirus-Adenovirus receptor during viral myocarditis. Cardiovasc Pathol 2008;17:81-92
-
(2008)
Cardiovasc Pathol
, vol.17
, pp. 81-92
-
-
Ruppert, V.1
Meyer, T.2
Pankuweit, S.3
Jonsdottir, T.4
Maisch, B.5
|