Myeloid-derived growth factor (C19orf10) mediates cardiac repair following myocardial infarction

Nature Medicine

Volumn 21, Issue 2, 2015, Pages 140-149

  Korf Klingebiel, Mortimer ^a   Reboll, Marc R ^a   Klede, Stefanie ^a   Brod, Torben ^a   Pich, Andreas ^a   Polten, Felix ^a   Napp, L Christian ^a   Bauersachs, Johann ^a   Ganser, Arnold ^a   Brinkmann, Eva ^a   Reimann, Ines ^a   Kempf, Tibor ^a   Niessen, Hans W ^b   Mizrahi, Jacques ^c   Schönfeld, Hans Joachim ^c   Iglesias, Antonio ^c   Bobadilla, Maria ^c   Wang, Yong ^a   Wollert, Kai C ^a  

^a HANNOVER MEDICAL SCHOOL   (Germany)

^b VU UNIVERSITY MEDICAL CENTER   (Netherlands)

^c F HOFFMANN LA ROCHE LTD   (Switzerland)

Author keywords

[No Author keywords available]

Indexed keywords

PROTEIN MYELOID DERIVED GROWTH FACTOR; RECOMBINANT PROTEIN; UNCLASSIFIED DRUG; C19ORF10 PROTEIN, HUMAN; D17WSU104E PROTEIN, MOUSE; INTERLEUKIN DERIVATIVE;

ACUTE HEART INFARCTION; ANGIOGENESIS; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BONE MARROW CELL; CELL DEATH; CELL PROLIFERATION; CELL PROTECTION; CELL SURVIVAL; CHROMOSOME 19; CONTINUOUS INFUSION; CONTROLLED STUDY; ENDOTHELIUM CELL; HEART INFARCTION; HEART MUSCLE CELL; HEART PROTECTION; HUMAN; HUMAN CELL; HUMAN TISSUE; MACROPHAGE; MALE; MONOCYTE; MOUSE; NONHUMAN; OPEN READING FRAME; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN SECRETION; RAT; TISSUE REPAIR; WILD TYPE; ANIMAL; DRUG EFFECTS; GENETICS; HEART VENTRICLE REMODELING; HEK293 CELL LINE; KNOCKOUT MOUSE; METABOLISM; REPERFUSION INJURY; SECRETION (PROCESS);

ANIMALS; BONE MARROW CELLS; CELL PROLIFERATION; ENDOTHELIAL CELLS; HEK293 CELLS; HUMANS; INTERLEUKINS; MACROPHAGES; MICE; MICE, KNOCKOUT; MONOCYTES; MYOCARDIAL INFARCTION; MYOCARDIAL REPERFUSION INJURY; MYOCYTES, CARDIAC; NEOVASCULARIZATION, PHYSIOLOGIC; VENTRICULAR REMODELING;

EID: 84922609359     PISSN: 10788956     EISSN: 1546170X     Source Type: Journal    
DOI: 10.1038/nm.3778     Document Type: Article

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