Toll-like receptor 5 deficiency exacerbates cardiac injury and inflammation induced by myocardial ischaemia-reperfusion in the mouse

Clinical Science

Volumn 129, Issue 2, 2015, Pages 187-198

  Parapanov, Roumen ^a   Lugrin, Jéřome ^a   Rosenblatt Velin, Nathalie ^a   Feihl, François ^a   Waeber, Bernard ^a   Milano, Giuseppina ^a   Vergely, Catherine ^b   Li, Na ^b   Pacher, Pal ^c   Liaudet, Lucas ^a  

^a LAUSANNE UNIVERSITY HOSPITAL   (Switzerland)

^b UNIVERSITÉ DE BOURGOGNE   (France)

^c NATIONAL INSTITUTES OF HEALTH   (United States)

Author keywords

Animal model; Inflammation; Innate immunity; Myocardial infarction; Oxidative stress; Toll like receptors

Indexed keywords

MITOGEN ACTIVATED PROTEIN KINASE; PROTEIN KINASE B; TOLL LIKE RECEPTOR 2; TOLL LIKE RECEPTOR 3; TOLL LIKE RECEPTOR 4; TOLL LIKE RECEPTOR 5; AUTACOID; MITOGEN ACTIVATED PROTEIN KINASE P38;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONDUCTANCE; CONTROLLED STUDY; DISEASE EXACERBATION; ECHOGRAPHY; ENZYME PHOSPHORYLATION; HEART FUNCTION; HEART INFARCTION SIZE; HEART INJURY; HEART MUSCLE INJURY; HEART MUSCLE ISCHEMIA; MALE; MORPHOLOGY; MOUSE; MYOCARDITIS; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN DEFICIENCY; PROTEIN EXPRESSION; TOLL LIKE RECEPTOR 5 DEFICIENCY; VOLUMETRY; ANIMAL; C57BL MOUSE; DEFICIENCY; DISEASE MODEL; GENETICS; GENOTYPE; HEART INFARCTION; HEART LEFT VENTRICLE FUNCTION; HEART MUSCLE; IMMUNOLOGY; INFLAMMATION; INNATE IMMUNITY; KNOCKOUT MOUSE; METABOLISM; PATHOLOGY; PATHOPHYSIOLOGY; PHENOTYPE; PHOSPHORYLATION; REPERFUSION INJURY;

ANIMALS; DISEASE MODELS, ANIMAL; GENOTYPE; IMMUNITY, INNATE; INFLAMMATION; INFLAMMATION MEDIATORS; MALE; MICE, INBRED C57BL; MICE, KNOCKOUT; MYOCARDIAL INFARCTION; MYOCARDIAL REPERFUSION INJURY; MYOCARDIUM; OXIDATIVE STRESS; P38 MITOGEN-ACTIVATED PROTEIN KINASES; PHENOTYPE; PHOSPHORYLATION; PROTO-ONCOGENE PROTEINS C-AKT; TOLL-LIKE RECEPTOR 5; VENTRICULAR DYSFUNCTION, LEFT; VENTRICULAR FUNCTION, LEFT;

EID: 84935510726     PISSN: 01435221     EISSN: 14708736     Source Type: Journal    
DOI: 10.1042/CS20140444     Document Type: Article

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