An emerging role for misfolded wild-type SOD1 in sporadic ALS pathogenesis

Frontiers in Cellular Neuroscience

Volumn 7, Issue DEC, 2013, Pages

  Rotunno, Melissa S ^a   Bosco, Daryl A ^a  

^a UNIVERSITY OF MASSACHUSETTS MEDICAL SCHOOL   (United States)

Author keywords

Amyotrophic lateral sclerosis (ALS); Immunotherapy; Protein misfolding; SOD1; Sporadic amyotrophic lateral sclerosis

Indexed keywords

COPPER ZINC SUPEROXIDE DISMUTASE; MITOGEN ACTIVATED PROTEIN KINASE 1; MITOGEN ACTIVATED PROTEIN KINASE 3; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE OXIDASE 2;

AMYOTROPHIC LATERAL SCLEROSIS; CALCIUM HOMEOSTASIS; DISEASE COURSE; ELECTROSPRAY; HYDROPHOBIC INTERACTION CHROMATOGRAPHY; IMMUNOREACTIVITY; MASS SPECTROMETRY; NERVE FIBER TRANSPORT; NONHUMAN; NUCLEAR MAGNETIC RESONANCE SPECTROSCOPY; PROTEIN FOLDING; PROTEIN PHOSPHORYLATION; REVIEW; SIGNAL TRANSDUCTION;

EID: 84890890208     PISSN: 16625102     EISSN: None     Source Type: Journal    
DOI: 10.3389/fncel.2013.00253     Document Type: Review

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