Acinar cell reprogramming: A clinically important target in pancreatic disease

Epigenomics

Volumn 7, Issue 2, 2015, Pages 267-281

  Pin, Christopher L ^a,b   Ryan, Joanna F ^a,b   Mehmood, Rashid ^a,b  

^a UNIVERSITY OF WESTERN ONTARIO   (Canada)

^b CHILDREN S HEALTH RESEARCH INSTITUTE   (Canada)

Author keywords

acinar to duct cell metaplasia; MIST1; NOTCH; pancreatic cancer; pancreatitis; polycomb repressor complex 2; Type I diabetes

Indexed keywords

TRANSCRIPTION FACTOR;

ACINAR CELL; CELL DIFFERENTIATION; ENDOCRINE CELL; EPIGENETICS; GENETIC REGULATION; HUMAN; INSULIN DEPENDENT DIABETES MELLITUS; METAPLASIA; NONHUMAN; PANCREAS ADENOCARCINOMA; PANCREAS ADENOMA; PANCREAS DISEASE; PANCREAS DUCT; PANCREAS ISLET BETA CELL; PHENOTYPE; PRIORITY JOURNAL; PROTEIN EXPRESSION; REVIEW; ANIMAL; CYTOLOGY; DIABETES MELLITUS, TYPE 1; EMBRYOLOGY; GENETIC EPIGENESIS; METABOLISM; MOUSE; NUCLEAR REPROGRAMMING; PANCREAS; PANCREATIC NEOPLASMS;

ACINAR CELLS; ANIMALS; CELL DIFFERENTIATION; CELLULAR REPROGRAMMING; DIABETES MELLITUS, TYPE 1; EPIGENESIS, GENETIC; HUMANS; INSULIN-SECRETING CELLS; MICE; PANCREAS; PANCREATIC DISEASES; PANCREATIC DUCTS; PANCREATIC NEOPLASMS;

EID: 84928881465     PISSN: 17501911     EISSN: 1750192X     Source Type: Journal    
DOI: 10.2217/epi.14.83     Document Type: Review

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