Analysis of the human monocyte-derived macrophage transcriptome and response to lipopolysaccharide provides new insights into genetic aetiology of inflammatory bowel disease

PLoS Genetics

Volumn 13, Issue 3, 2017, Pages

  Baillie, J Kenneth ^a   Arner, Erik ^b   Daub, Carsten ^b   De Hoon, Michiel ^b   Itoh, Masayoshi ^b   Kawaji, Hideya ^b   Lassmann, Timo ^b   Carninci, Piero ^b   Forrest, Alistair R R ^b   Hayashizaki, Yoshihide ^b   Faulkner, Geoffrey J ^a   Wells, Christine A ^c   Rehli, Michael ^d   Pavli, Paul ^e   Summers, Kim M ^a   Hume, David A ^a  

^a UNIVERSITY OF EDINBURGH   (United Kingdom)

^b RIKEN   (Japan)

^c UNIVERSITY OF QUEENSLAND   (Australia)

^d UNIVERSITY HOSPITAL REGENSBURG   (Germany)

^e AUSTRALIAN NATIONAL UNIVERSITY   (Australia)

Author keywords

[No Author keywords available]

Indexed keywords

COLONY STIMULATING FACTOR 1; LIPOPOLYSACCHARIDE; LONG UNTRANSLATED RNA; RNA; TRANSCRIPTOME; CYTOKINE; LIGAND; TRANSCRIPTION FACTOR;

ARTICLE; CELL ACTIVATION; CELL DIFFERENTIATION; CONTROLLED STUDY; ENHANCER REGION; GENE CLUSTER; GENE EXPRESSION; GENE LOCUS; GENOME-WIDE ASSOCIATION STUDY; HUMAN; HUMAN CELL; INFLAMMATORY BOWEL DISEASE; LAMINA PROPRIA; MACROPHAGE; MONOCYTE; PATHOGENESIS; PROMOTER REGION; RNA TRANSCRIPTION; TRANSCRIPTION INITIATION; CYTOLOGY; GENE EXPRESSION REGULATION; GENETIC PREDISPOSITION; GENETIC TRANSCRIPTION; GENETICS; GENOMICS; INFLAMMATION; INFLAMMATORY BOWEL DISEASES; INTESTINE MUCOSA; METABOLISM; MULTIGENE FAMILY; PROTEIN MOTIF; TIME FACTOR;

AMINO ACID MOTIFS; CELL DIFFERENTIATION; CYTOKINES; GENE EXPRESSION REGULATION; GENETIC PREDISPOSITION TO DISEASE; GENOME-WIDE ASSOCIATION STUDY; GENOMICS; HUMANS; INFLAMMATION; INFLAMMATORY BOWEL DISEASES; INTESTINAL MUCOSA; LIGANDS; LIPOPOLYSACCHARIDES; MACROPHAGE COLONY-STIMULATING FACTOR; MACROPHAGES; MONOCYTES; MULTIGENE FAMILY; PROMOTER REGIONS, GENETIC; TIME FACTORS; TRANSCRIPTION FACTORS; TRANSCRIPTION, GENETIC; TRANSCRIPTIONAL ACTIVATION; TRANSCRIPTOME;

EID: 85016560823     PISSN: 15537390     EISSN: 15537404     Source Type: Journal    
DOI: 10.1371/journal.pgen.1006641     Document Type: Article

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