Hypothesis-free analysis of ATG16L1 demonstrates gene-wide extent of association with Crohn's disease susceptibility

Gut

Volumn 62, Issue 2, 2013, Pages 331-333

  Van Limbergen, Johan ^a,b,c   Kabakchiev, Boyko ^d   Stempak, Joanne M ^d   Schumm, Phil ^e   Xu, Wei ^f   Henderson, Paul ^a   Girardin, Stephen ^c   Griffiths, Anne M ^b   Philpott, Dana ^b   Silverberg, Mark ^d  

^a UNIVERSITY OF EDINBURGH   (United Kingdom)

^b HOSPITAL FOR SICK CHILDREN   (Canada)

^c UNIVERSITY OF TORONTO   (Canada)

^d MOUNT SINAI HOSPITAL   (Canada)

^e UNIVERSITY OF CHICAGO   (United States)

^f UNIVERSITY OF TORONTO   (Canada)

Author keywords

[No Author keywords available]

Indexed keywords

ALANINE; CYTOKINE; THREONINE;

3' UNTRANSLATED REGION; 5' UNTRANSLATED REGION; AUTOPHAGY; CROHN DISEASE; CYTOKINE PRODUCTION; DISEASE ASSOCIATION; DISEASE PREDISPOSITION; EXON; GENE FREQUENCY; GENE LINKAGE DISEQUILIBRIUM; GENE LOCUS; GENE SEQUENCE; GENETIC ASSOCIATION; GENOTYPE; HUMAN; INTRON; LETTER; MUTATIONAL ANALYSIS; PRIORITY JOURNAL; SINGLE NUCLEOTIDE POLYMORPHISM;

CARRIER PROTEINS; CROHN DISEASE; CYTOKINES; HUMANS; INFLAMMATION MEDIATORS; NOD2 SIGNALING ADAPTOR PROTEIN;

EID: 84872053666     PISSN: 00175749     EISSN: 14683288     Source Type: Journal    
DOI: 10.1136/gutjnl-2012-303165     Document Type: Letter

References (8)

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