Animal models of colorectal cancer

Cancer and Metastasis Reviews

Volumn 32, Issue 1-2, 2013, Pages 39-61

  Johnson, Robert L ^a   Fleet, James C ^b,c  

^a PURDUE UNIVERSITY   (United States)

^b PURDUE UNIVERSITY   (United States)

^c Purdue University   (United States)

Author keywords

Chemically induced; Genetically modified mice; Heritable; Sporadic

Indexed keywords

APC PROTEIN; B RAF KINASE; BETA CATENIN; CALCIUM; CYCLIN DEPENDENT KINASE 8; CYCLIN DEPENDENT KINASE INHIBITOR 1; EPIDERMAL GROWTH FACTOR RECEPTOR; EPIDERMAL GROWTH FACTOR RECEPTOR 2; GROWTH FACTOR RECEPTOR; K RAS PROTEIN; MITOGEN ACTIVATED PROTEIN KINASE; PHOSPHATIDYLINOSITOL 3 KINASE; PHOSPHATIDYLINOSITOL 3,4,5 TRISPHOSPHATE; PHOSPHATIDYLINOSITOL 3,4,5 TRISPHOSPHATE 3 PHOSPHATASE; PROTEIN BAX; PROTEIN KINASE B; PROTEIN MLH1; PROTEIN MSH2; PROTEIN MSH6; PROTEIN P53; RAS PROTEIN; SMAD PROTEIN; SMAD2 PROTEIN; SMAD3 PROTEIN; SMAD4 PROTEIN; TRANSFORMING GROWTH FACTOR BETA; TRANSFORMING GROWTH FACTOR BETA RECEPTOR 2; UNINDEXED DRUG; VITAMIN D; WNT PROTEIN;

APOPTOSIS; ARTICLE; ASCENDING COLON; CANCER EPIDEMIOLOGY; CANCER INCIDENCE; CELL SURVIVAL; CHROMOSOMAL INSTABILITY; CLINICAL FEATURE; COLON CANCER; COLORECTAL CANCER; DESCENDING COLON; DIET SUPPLEMENTATION; DOG; FAMILIAL COLON POLYPOSIS; GENE DELETION; GENE EXPRESSION; GENETIC VARIABILITY; HISTOPATHOLOGY; HUMAN; IN VIVO STUDY; MISMATCH REPAIR; MOLECULAR GENETICS; NONHUMAN; PATHOGENESIS; PHENOTYPE; PRIORITY JOURNAL; PROMOTER REGION; RISK FACTOR; RODENT; SOMATIC MUTATION; TUMOR SUPPRESSOR GENE; TUMOR XENOGRAFT;

ANIMALS; COLORECTAL NEOPLASMS; DISEASE MODELS, ANIMAL; HUMANS; NEOPLASM METASTASIS; PHENOTYPE;

EID: 84878114332     PISSN: 01677659     EISSN: 15737233     Source Type: Journal    
DOI: 10.1007/s10555-012-9404-6     Document Type: Article

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