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Volumn 698, Issue 1-3, 2013, Pages 6-18

Excitotoxicity: Bridge to various triggers in neurodegenerative disorders

Author keywords

Calcium overload; Excitotoxicity; Glutamate; Mitochondrial dysfunction; Neurodegenerative diseases; Oxidative stress; Therapeutics

Indexed keywords

4 AMINOBUTYRIC ACID A RECEPTOR; ADENOSINE TRIPHOSPHATE; CALCIUM; CHLORINE; COGANE; COLECALCIFEROL; CURCUMIN; FARNOQUINONE; GLUTAMATE RECEPTOR; GLUTAMATE RECEPTOR ANTAGONIST; GLUTAMIC ACID; HYDROXYMETHYLGLUTARYL COENZYME A REDUCTASE INHIBITOR; ICOSANOID; LIPOXYGENASE; METFORMIN; N METHYL DEXTRO ASPARTIC ACID RECEPTOR; N METHYL DEXTRO ASPARTIC ACID RECEPTOR BLOCKING AGENT; NEUROTRANSMITTER; NITRIC OXIDE; NOOTROPIC AGENT; PERAMPANEL; PHOSPHODIESTERASE; PHOSPHOLIPASE A2; PROSTAGLANDIN E2; PROSTAGLANDIN SYNTHASE; REACTIVE OXYGEN METABOLITE; RESVERATROL; TENECTEPLASE; UNCLASSIFIED DRUG; UNINDEXED DRUG; VINIFERIN;

EID: 84871919598     PISSN: 00142999     EISSN: 18790712     Source Type: Journal    
DOI: 10.1016/j.ejphar.2012.10.032     Document Type: Review
Times cited : (529)

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