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Volumn 654, Issue , 2010, Pages 421-445

Molecular pathways underlying the pathogenesis of pancreatic α-cell dysfunction

Author keywords

cells; Counter regulation; Development; Diabetes; GABA; GLP 1; Glucagon; Hypertrophy; Hypoglycemia; Insulin; Neurotransmitters and nervous systems; Pancreatic islets; Secretion; Somatostatin; Zinc

Indexed keywords

4 AMINOBUTYRIC ACID; ANTISENSE OLIGONUCLEOTIDE; CYCLIC AMP; CYCLIC AMP DEPENDENT PROTEIN KINASE; EXENDIN 4; GHRELIN; GLITAZONE DERIVATIVE; GLUCAGON; GLUCAGON LIKE PEPTIDE 1; GLUCAGON LIKE PEPTIDE 2; GLUCAGON RECEPTOR; GLUCOSE TRANSPORTER 2; HEPATOCYTE NUCLEAR FACTOR 3BETA; INSULIN; ION CHANNEL; LIRAGLUTIDE; NEUROGENIC DIFFERENTIATION FACTOR; OXYNTOMODULIN; PIOGLITAZONE; ROSIGLITAZONE; SOMATOSTATIN; TRANSCRIPTION FACTOR CDX2; TRANSCRIPTION FACTOR MAFA; TRANSCRIPTION FACTOR MAFB; TRANSCRIPTION FACTOR NKX6.1; TRANSCRIPTION FACTOR PAX4; TRANSCRIPTION FACTOR PAX6; VILDAGLIPTIN; ZINC; AGENTS INTERACTING WITH TRANSMITTER, HORMONE OR DRUG RECEPTORS;

EID: 77953939472     PISSN: 00652598     EISSN: None     Source Type: Book Series    
DOI: 10.1007/978-90-481-3271-3_18     Document Type: Article
Times cited : (26)

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