Excitotoxicity in ALS: Overstimulation, or overreaction?

Experimental Neurology

Volumn 275, Issue , 2016, Pages 162-171

  King, Anna E ^a   Woodhouse, Adele ^a   Kirkcaldie, Matthew T K ^a   Vickers, James C ^a  

^a UNIVERSITY OF TASMANIA   (Australia)

Author keywords

ALS; EAAT2; Excitability; GABA; Glutamate; Glycine; Interneuron; Motor neuron disease

Indexed keywords

GLUTAMATE RECEPTOR; GLUTAMIC ACID;

AMYOTROPHIC LATERAL SCLEROSIS; CALCIUM CELL LEVEL; EXCITOTOXICITY; GABAERGIC TRANSMISSION; GLUTAMATERGIC SYNAPSE; HUMAN; INTERNEURON; MOLECULAR PATHOLOGY; NERVE EXCITABILITY; NEUROPATHOLOGY; NEUROTRANSMITTER RELEASE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN SECRETION; RECEPTOR INTRINSIC ACTIVITY; REVIEW; SYNAPTIC TRANSMISSION; CELL DEATH; METABOLISM; MOTONEURON; NERVE DEGENERATION; PATHOLOGY; PHYSIOLOGY; SPINAL CORD;

AMYOTROPHIC LATERAL SCLEROSIS; CELL DEATH; GLUTAMIC ACID; HUMANS; MOTOR NEURONS; NERVE DEGENERATION; SPINAL CORD;

EID: 84946893765     PISSN: 00144886     EISSN: 10902430     Source Type: Journal    
DOI: 10.1016/j.expneurol.2015.09.019     Document Type: Review

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