Motor neuron degeneration induced by excitotoxin agonists has features in common with those seen in the SOD-1 transgenic mouse model of amyotrophic lateral sclerosis

Journal of Neuropathology and Experimental Neurology

Volumn 55, Issue 2, 1996, Pages 211-224

  Ikonomidou, Chrysanthy ^a   Qin, Yue Qin ^a   Labruyere, Joann ^a   Olney, John W ^a  

^a WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

AMPA; Amyotrophic lateral sclerosis; DL homocysteic acid; Excitotoxicity; Kainic acid; NMDA; SOD 1 transgenic mouse

Indexed keywords

AMPA RECEPTOR; COPPER ZINC SUPEROXIDE DISMUTASE; EXCITATORY AMINO ACID RECEPTOR; GLUTAMATE RECEPTOR; HOMOCYSTEIC ACID; KAINIC ACID; KAINIC ACID RECEPTOR; N METHYL DEXTRO ASPARTIC ACID; QUISQUALIC ACID;

AMYOTROPHIC LATERAL SCLEROSIS; ANIMAL CELL; ARTICLE; FAMILIAL DISEASE; LUMBAR SPINAL CORD; MOTOR NEURON DISEASE; NERVE DEGENERATION; NEUROTRANSMISSION; NEWBORN; NONHUMAN; PATHOGENESIS; PRIORITY JOURNAL; RAT; TRANSGENIC MOUSE;

EID: 13344261949     PISSN: 00223069     EISSN: None     Source Type: Journal    
DOI: 10.1097/00005072-199602000-00010     Document Type: Article

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