Neuronal and glial glutamate transporters possess an SH-based redox regulatory mechanism

European Journal of Neuroscience

Volumn 9, Issue 6, 1997, Pages 1236-1243

  Trotti, Davide ^a   Rizzini, Barbara Lodi ^a   Rossi, Daniela ^a   Haugeto, Oyvind ^b   Racagni, Giorgio ^a   Danbolt, Niels C ^b   Volterra, Andrea ^a  

^a UNIVERSITY OF MILAN   (Italy)

^b UNIVERSITY OF OSLO   (Norway)

Author keywords

Endogenous oxidants and reductants; Excitatory neurotransmission; Excitotoxicity; GLT1 and GLAST transporters; Oxidative stress; Rat EAAC1

Indexed keywords

5,5' DITHIOBIS(2 NITROBENZOIC ACID); CARRIER PROTEIN; CYSTEINE; DISULFIDE; DITHIOTHREITOL; GLUTAMIC ACID; HYDROGEN PEROXIDE; LIPOSOME; RECOMBINANT PROTEIN; THIOL;

ARTICLE; ASTROCYTE; CELL CULTURE; GLIA CELL; NERVE CELL; NEUROTRANSMISSION; OXIDATION; OXIDATION REDUCTION REACTION; PATHOPHYSIOLOGY; PRIORITY JOURNAL;

EID: 0031001036     PISSN: 0953816X     EISSN: None     Source Type: Journal    
DOI: 10.1111/j.1460-9568.1997.tb01478.x     Document Type: Article

References (2)

1. Aizenman, E., Lipton, S. A. and Loring, R. H. (1989) Selective modulation of NMDA responses by reduction and oxidation. Neuron, 2,1257-1263.
2. Aizenman, E., Hartnett, K. A. and Reynolds, I. J. (1990) Oxygen free radicals regulate NMDA receptor function via a redox modulatory site. Neuron, 5, 841-846.