Early pathogenesis in the adult-onset neurodegenerative disease amyotrophic lateral sclerosis

Journal of Cellular Biochemistry

Volumn 113, Issue 11, 2012, Pages 3301-3312

  Van Zundert, Brigitte ^a   Izaurieta, Pamela ^a,b   Fritz, Elsa ^a,b   Alvarez, Francisco J ^c  

^a UNIVERSIDAD ANDRÉS BELLO   (Chile)

^b UNIVERSITY OF CONCEPCIÓN   (Chile)

^c EMORY UNIVERSITY   (United States)

Author keywords

ALS; COMPENSATION; EXCITOTOXICITY; PATHOLOGY; SODIUM CHANNELS; SYNAPSE

Indexed keywords

COPPER ZINC SUPEROXIDE DISMUTASE; EXCITATORY AMINO ACID TRANSPORTER 2; GLUCOSE TRANSPORTER 1; GLUTAMIC ACID; N METHYL DEXTRO ASPARTIC ACID RECEPTOR 2A; N METHYL DEXTRO ASPARTIC ACID RECEPTOR 2B; PROTEIN P40; PROTEIN P50; RAC1 PROTEIN; REACTIVE NITROGEN SPECIES; REACTIVE OXYGEN METABOLITE; SODIUM CHANNEL NAV1.1; SODIUM CHANNEL NAV1.2; SODIUM CHANNEL NAV1.3; SODIUM CHANNEL NAV1.6;

ADULTHOOD; AGING; AMYOTROPHIC LATERAL SCLEROSIS; APOPTOSIS; ARTICLE; ASTROCYTE; BEHAVIOR DISORDER; CALCIUM HOMEOSTASIS; CELL DEATH; CYTOSKELETON; DEGENERATIVE DISEASE; DISEASE COURSE; DNA DAMAGE; ENDOPLASMIC RETICULUM STRESS; EXCITOTOXICITY; GENE MUTATION; HUMAN; MOTONEURON; NERVE CELL EXCITABILITY; NERVE CELL LESION; NERVE FIBER TRANSPORT; NONHUMAN; ONSET AGE; OXIDATIVE STRESS; PATHOGENESIS; PHENOTYPE; PRIORITY JOURNAL; SODIUM CURRENT; SYMPTOM; UNFOLDED PROTEIN RESPONSE;

AMYOTROPHIC LATERAL SCLEROSIS; ANIMALS; ANIMALS, NEWBORN; CALCIUM; DISEASE MODELS, ANIMAL; HUMANS; MICE; MICE, TRANSGENIC; MOTOR NEURONS; MUTATION; SODIUM CHANNELS; SUPEROXIDE DISMUTASE; SYNAPSES;

MUS; MUS MUSCULUS;

EID: 84866483742     PISSN: 07302312     EISSN: 10974644     Source Type: Journal    
DOI: 10.1002/jcb.24234     Document Type: Article

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