Increased persistent Na+ current and its effect on excitability in motoneurones cultured from mutant SOD1 mice

Journal of Physiology

Volumn 563, Issue 3, 2005, Pages 843-854

  Kuo, J J ^a   Siddique, T ^a   Fu, R ^a   Heckman, C J ^a  

^a NORTHWESTERN UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ALANINE; COPPER ZINC SUPEROXIDE DISMUTASE; GLYCINE; RILUZOLE; SODIUM;

AMINO ACID SUBSTITUTION; AMYOTROPHIC LATERAL SCLEROSIS; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BASE PAIRING; CONTROLLED STUDY; DRUG MECHANISM; GENE MUTATION; MOTONEURON; MOUSE; MOUSE MUTANT; NERVE CELL CULTURE; NERVE CELL EXCITABILITY; NERVE CELL MEMBRANE CONDUCTANCE; NONHUMAN; PATHOGENESIS; PRIORITY JOURNAL; SODIUM CURRENT; CELL MEMBRANE CONDUCTANCE; CONCENTRATION RESPONSE; EMBRYO; GENE OVEREXPRESSION; NERVE CELL DEGENERATION; SPINAL CORD MOTONEURON; SYMPTOM; TRANSGENIC MOUSE;

ACTION POTENTIALS; ADAPTATION, PHYSIOLOGICAL; ANIMALS; CELLS, CULTURED; ION CHANNEL GATING; MEMBRANE POTENTIALS; MICE; MICE, TRANSGENIC; MOTOR NEURONS; MUTAGENESIS, SITE-DIRECTED; MUTATION; NEURONAL PLASTICITY; SODIUM; SODIUM CHANNELS; SPINAL CORD; SUPEROXIDE DISMUTASE;

EID: 15844431408     PISSN: 00223751     EISSN: None     Source Type: Journal    
DOI: 10.1113/jphysiol.2004.074138     Document Type: Article

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