How cancer cells hijack DNA double-strand break repair pathways to gain genomic instability

Biochemical Journal

Volumn 471, Issue 1, 2015, Pages 1-11

  Jeggo, Penny A ^a   Löbrichf, Markus ^b  

^a UNIVERSITY OF SUSSEX   (United Kingdom)

^b DARMSTADT UNIVERSITY OF TECHNOLOGY   (Germany)

Author keywords

Alternative NHEJ; Cancer cells; Double strand break repair; Genomic stability; Homologous recombination; Non homologous end joining

Indexed keywords

DOUBLE STRANDED DNA;

ARTICLE; CANCER CELL; CELL CYCLE CHECKPOINT; CHROMATIN ASSEMBLY AND DISASSEMBLY; DNA END JOINING REPAIR; DNA STRAND BREAKAGE; GENOMIC INSTABILITY; HOMOLOGOUS RECOMBINATION; HUMAN; NONHUMAN; PRIORITY JOURNAL; SIGNAL TRANSDUCTION; ANIMAL; DNA REPAIR; DOUBLE STRANDED DNA BREAK; GENETICS; METABOLISM; NEOPLASM; PATHOLOGY;

ANIMALS; CELL CYCLE CHECKPOINTS; DNA BREAKS, DOUBLE-STRANDED; DNA REPAIR; GENOMIC INSTABILITY; HUMANS; NEOPLASMS; SIGNAL TRANSDUCTION;

EID: 84942342661     PISSN: 02646021     EISSN: 14708728     Source Type: Journal    
DOI: 10.1042/BJ20150582     Document Type: Article

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