The cell biology of prion-like spread of protein aggregates: Mechanisms and implication in neurodegeneration

Biochemical Journal

Volumn 452, Issue 1, 2013, Pages 1-17

  Costanzo, Maddalena ^a   Zurzolo, Chiara ^a  

^a INSTITUT PASTEUR   (France)

Author keywords

A synuclein; Amyloid ; Cell to cell transfer; Huntingtin; Prion like; Tau

Indexed keywords

ALPHA SYNUCLEIN; AMYLOID BETA PROTEIN; CASPASE 3; HUNTINGTIN; PRION PROTEIN; TAU PROTEIN;

ALZHEIMER DISEASE; CELL CONTACT; CELL SPREADING; CYTOSOL; DISEASE COURSE; ENDOCYTOSIS; ENDOPLASMIC RETICULUM; EXOSOME; HUMAN; HUNTINGTON CHOREA; INTERNALIZATION; LEWY BODY; NEUROPATHOLOGY; NEUROTOXICITY; PARKINSON DISEASE; PRIORITY JOURNAL; PROTEIN AGGREGATION; PROTEIN CONFORMATION; PROTEIN FOLDING; PROTEIN LOCALIZATION; PROTEIN SECRETION; PROTEIN TRANSPORT; REVIEW; SYNAPTIC TRANSMISSION;

ANIMALS; CELL COMMUNICATION; HUMANS; NEURODEGENERATIVE DISEASES; PRION DISEASES; PRIONS; PROTEIN FOLDING; PROTEIN STRUCTURE, TERTIARY; PROTEIN TRANSPORT;

EID: 84876945450     PISSN: 02646021     EISSN: 14708728     Source Type: Journal    
DOI: 10.1042/BJ20121898     Document Type: Review

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