Senescence in Health and Disease

Cell

Volumn 169, Issue 6, 2017, Pages 1000-1011

  He, Shenghui ^a   Sharpless, Norman E ^a  

^a UNIVERSITY OF NORTH CAROLINA SCHOOL OF MEDICINE   (United States)

Author keywords

aging; cancer; cellular senescence; DNA damage; molecular age; p16INK4a; SASP; senolysis; telomere; tumor suppression

Indexed keywords

ANGIOGENESIS INHIBITOR; BIOLOGICAL MARKER; CORTICOSTEROID; CYCLIN DEPENDENT KINASE INHIBITOR 2A; CYTOKINE; CYTOTOXIC AGENT; GROWTH FACTOR; NAVITOCLAX; PROTEIN P53; RETINOBLASTOMA PROTEIN; TELOMERASE; VENETOCLAX;

APOPTOSIS; ATHEROSCLEROSIS; CANCER INHIBITION; CANCER RISK; CELL AGING; CELL CYCLE ARREST; CELL DAMAGE; CELL GROWTH; CELL MATURATION; CELL PROLIFERATION; CELL SURVIVAL; CELLULAR IMMUNITY; CELLULAR STRESS RESPONSE; CHRONOLOGY; FATIGUE; GLAUCOMA; GROWTH DISORDER; HEALTH; HISTOPATHOLOGY; HOST CELL; HUMAN; IN VIVO STUDY; MALIGNANT NEOPLASM; MORBIDITY; NON INSULIN DEPENDENT DIABETES MELLITUS; NONHUMAN; OBESITY; OXIDATIVE STRESS; PRIORITY JOURNAL; REVIEW; RISK FACTOR; SARCOPENIA; WOUND HEALING; AGING; ANIMAL; CELL CYCLE; IMMUNOLOGY; NEOPLASM; PATHOLOGY;

AGING; ANIMALS; CELL AGING; CELL CYCLE; HUMANS; NEOPLASMS; WOUND HEALING;

EID: 85020028464     PISSN: 00928674     EISSN: 10974172     Source Type: Journal    
DOI: 10.1016/j.cell.2017.05.015     Document Type: Review

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