Coupling endoplasmic reticulum stress to the cell death program

Cell Death and Differentiation

Volumn 11, Issue 4, 2004, Pages 372-380

  Rao, R V ^a   Ellerby, H M ^a   Bredesen, D E ^a,b  

^a BUCK INSTITUTE FOR RESEARCH ON AGING   (United States)

^b UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

Apoptosis; Apostat; Caspase; Endoplasmic reticulum; ER stress; Misfolded proteins

Indexed keywords

CALCIUM ION; CASPASE; CASPASE 8; CASPASE 9; CYSTEINE PROTEINASE; FAS ANTIGEN; TUMOR NECROSIS FACTOR ALPHA RECEPTOR;

APOPTOSIS; CALCIUM CELL LEVEL; CALCIUM HOMEOSTASIS; CELL COMPARTMENTALIZATION; CELL DEATH; CELL FUNCTION; CYTOPLASM; DEGENERATIVE DISEASE; ENDOPLASMIC RETICULUM; ENZYME LOCALIZATION; HUMAN; MITOCHONDRION; MOLECULAR DYNAMICS; NONHUMAN; PATHOGENESIS; PRIORITY JOURNAL; PROTEIN FOLDING; PROTEIN SYNTHESIS; PROTEIN TRANSPORT; REGULATORY MECHANISM; REVIEW; SIGNAL TRANSDUCTION; STRESS;

APOPTOSIS; CALCIUM; CASPASES; ENDOPLASMIC RETICULUM; MITOCHONDRIA; RECEPTORS, TUMOR NECROSIS FACTOR; SIGNAL TRANSDUCTION;

EID: 1842843860     PISSN: 13509047     EISSN: None     Source Type: Journal    
DOI: 10.1038/sj.cdd.4401378     Document Type: Review

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