Gadd153 sensitizes cells to endoplasmic reticulum stress by down-regulating Bc12 and perturbing the cellular redox state

Molecular and Cellular Biology

Volumn 21, Issue 4, 2001, Pages 1249-1259

  McCullough, K D ^a   Martindale, J L ^a   Klotz, L O ^a   Aw, T Y ^a   Holbrook, N J ^a  

^a Laboratory of Biological Chemistry   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

GLUTATHIONE; PROTEIN BCL 2; REACTIVE OXYGEN METABOLITE; TRANSCRIPTION FACTOR; TRANSCRIPTION FACTOR GADD153; UNCLASSIFIED DRUG;

APOPTOSIS; ARTICLE; CELL DEATH; CELL SURVIVAL; CONTROLLED STUDY; ENDOPLASMIC RETICULUM; FLOW CYTOMETRY; GENE OVEREXPRESSION; HELA CELL; HOMEOSTASIS; HUMAN; HUMAN CELL; OXIDATION REDUCTION REACTION; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN DEPLETION;

ANIMALS; BASE SEQUENCE; CCAAT-ENHANCER-BINDING PROTEINS; CELL LINE; DNA PRIMERS; DOWN-REGULATION; ENDOPLASMIC RETICULUM; GENE EXPRESSION; GENES, BCL-2; GLUTATHIONE; HELA CELLS; HUMANS; MICE; MUTAGENESIS, SITE-DIRECTED; OXIDATION-REDUCTION; OXIDATIVE STRESS; PROTO-ONCOGENE PROTEINS C-BCL-2; TRANSCRIPTION FACTOR CHOP; TRANSCRIPTION FACTORS;

EID: 0035144493     PISSN: 02707306     EISSN: None     Source Type: Journal    
DOI: 10.1128/MCB.21.4.1249-1259.2001     Document Type: Article

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