-
1
-
-
0033153086
-
The role of caspase in development, immunity, and apoptotic signal transduction: Lessons from knockout mice
-
Los, M., Wesselborg, S. & Schulze-Ostholff, K. The role of caspase in development, immunity, and apoptotic signal transduction: lessons from knockout mice. Immunity 10, 629-639 (1999).
-
(1999)
Immunity
, vol.10
, pp. 629-639
-
-
Los, M.1
Wesselborg, S.2
Schulze-Ostholff, K.3
-
2
-
-
0031730617
-
ICE, neuronal apoptosis and neurodegeneration
-
Friedland, R. M. & Yuan, J. ICE, neuronal apoptosis and neurodegeneration. Cell Death Differ. 5, 823-831 (1998).
-
(1998)
Cell Death Differ.
, vol.5
, pp. 823-831
-
-
Friedland, R.M.1
Yuan, J.2
-
3
-
-
2642689658
-
Proteases to die for
-
Cryns, V. & Yuan, J. Proteases to die for. Genes Dev. 12, 1551-1570 (1998).
-
(1998)
Genes Dev.
, vol.12
, pp. 1551-1570
-
-
Cryns, V.1
Yuan, J.2
-
4
-
-
15844412409
-
FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death inducing signaling complex
-
Muzio, M. et al. FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death inducing signaling complex. Cell 85, 817-827 (1996).
-
(1996)
Cell
, vol.85
, pp. 817-827
-
-
Muzio, M.1
-
5
-
-
0030011398
-
Involvement of MACH, a novel MORT1/ FADD-interacting protease, in Fas/APO-1-and TNF receptor-induced cell death
-
Boldin, M. P., Goncharov, T. M., Goltsev, Y. V. & Wallach, D. Involvement of MACH, a novel MORT1/ FADD-interacting protease, in Fas/APO-1-and TNF receptor-induced cell death. Cell 85, 803-815 (1996).
-
(1996)
Cell
, vol.85
, pp. 803-815
-
-
Boldin, M.P.1
Goncharov, T.M.2
Goltsev, Y.V.3
Wallach, D.4
-
6
-
-
0030715323
-
Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade
-
Li, P. et al., Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade. Cell 91, 479-489 (1997).
-
(1997)
Cell
, vol.91
, pp. 479-489
-
-
Li, P.1
-
7
-
-
0032780022
-
The cellular response to protein misfolding in the endoplasmic reticulum
-
Weilhinda, A. A., Tirasophon, W. & Kaufman, R. J. The cellular response to protein misfolding in the endoplasmic reticulum. Gene Expr. 7, 293-300 (1999).
-
(1999)
Gene Expr.
, vol.7
, pp. 293-300
-
-
Weilhinda, A.A.1
Tirasophon, W.2
Kaufman, R.J.3
-
8
-
-
0031042304
-
Characterization of seven murine caspase family members
-
Van de Craen, M. et al. Characterization of seven murine caspase family members. FEBS Lett 403, 61-69 (1997).
-
(1997)
FEBS Lett
, vol.403
, pp. 61-69
-
-
Van De Craen, M.1
-
9
-
-
0023852783
-
The presence of malfolded proteins in the endoplasmic reticulum signals the induction of glucose regulated proteins
-
Kozutsumi, Y., Segal, M., Normington, K., Gething, M. J. & Sambrook, J. The presence of malfolded proteins in the endoplasmic reticulum signals the induction of glucose regulated proteins. Nature 332, 462-464 (1988).
-
(1988)
Nature
, vol.332
, pp. 462-464
-
-
Kozutsumi, Y.1
Segal, M.2
Normington, K.3
Gething, M.J.4
Sambrook, J.5
-
10
-
-
8044231311
-
The alzheimer's disease-associated presenilins are differentially phosphorylatedproteins located predominantly within the endoplasmic reticulum
-
Walter, J. et al. The Alzheimer's disease-associated presenilins are differentially phosphorylatedproteins located predominantly within the endoplasmic reticulum. Mol. Med. 2, 673-691 (1996).
-
(1996)
Mol. Med.
, vol.2
, pp. 673-691
-
-
Walter, J.1
-
11
-
-
0028200649
-
Inhibition of energy metabolism alters the processing of amyloid precursor protein and induces a potentially amyloidogenic derivative
-
Gabuzda, D., Busciglio, J., Chen, L. B., Matsudaira, P. & Yankner, B. A. Inhibition of energy metabolism alters the processing of amyloid precursor protein and induces a potentially amyloidogenic derivative. J. Biol. Chem. 269, 13623-13628 (1994).
-
(1994)
J. Biol. Chem.
, vol.269
, pp. 13623-13628
-
-
Gabuzda, D.1
Busciglio, J.2
Chen, L.B.3
Matsudaira, P.4
Yankner, B.A.5
-
12
-
-
0032054744
-
CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulum
-
Zinszner, H. et al. CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulum. Gene Dev. 12, 982-995 (1998).
-
(1998)
Gene Dev.
, vol.12
, pp. 982-995
-
-
Zinszner, H.1
-
13
-
-
0024990330
-
Neurotrophic and neurotoxic effects of amyloid beta protein: Reversal by tachykinin neuropeptides
-
Yankner, B. A., Duffy, L. K. & Kirschner, D. A. Neurotrophic and neurotoxic effects of amyloid beta protein: reversal by tachykinin neuropeptides. Science 250, 279-282 (1990).
-
(1990)
Science
, vol.250
, pp. 279-282
-
-
Yankner, B.A.1
Duffy, L.K.2
Kirschner, D.A.3
-
14
-
-
0013079948
-
An intracellular protein that binds amyloid-beta peptide and mediates neurotoxicity in Alzheimer's disease
-
Yan, S. D. et al. An intracellular protein that binds amyloid-beta peptide and mediates neurotoxicity in Alzheimer's disease. Nature 389, 689-695 (1997).
-
(1997)
Nature
, vol.389
, pp. 689-695
-
-
Yan, S.D.1
-
15
-
-
0032548919
-
Murine caspase-11, an ICE interacting protease, is essential for the activation of ICE
-
Wang, S. et al. Murine caspase-11, an ICE interacting protease, is essential for the activation of ICE. Cell. 92, 501-509 (1998).
-
(1998)
Cell
, vol.92
, pp. 501-509
-
-
Wang, S.1
-
16
-
-
0030071721
-
Downregulation of Cu/Zn superoxide dismutase leads to cell death via the nitric oxide-peroxynitrite pathway
-
Troy, C. M., Derossi, D., Prochiantz, A., Greene, I., A. & Shelanski, M. L. Downregulation of Cu/Zn superoxide dismutase leads to cell death via the nitric oxide-peroxynitrite pathway. J. Neurosci. 16, 253-261 (1996).
-
(1996)
J. Neurosci.
, vol.16
, pp. 253-261
-
-
Troy, C.M.1
Derossi, D.2
Prochiantz, A.3
Greene, I.A.4
Shelanski, M.L.5
-
17
-
-
13344250473
-
Ataxia and epileptic seizures in mice lacking type I inositol 1,4-5-trisphosphate receptor
-
Matsumoto, M. et al. Ataxia and epileptic seizures in mice lacking type I inositol 1,4-5-trisphosphate receptor. Nature 379, 168-171 (1996).
-
(1996)
Nature
, vol.379
, pp. 168-171
-
-
Matsumoto, M.1
|