Amyotrophic lateral sclerosis: Unfolding the toxicity of the misfolded

Cell

Volumn 104, Issue 4, 2001, Pages 581-591

  Julien, Jean Pierre ^a  

^a MONTREAL GENERAL HOSPITAL   (Canada)

Author keywords

[No Author keywords available]

Indexed keywords

BENZYLOXYCARBONYLVALYLALANYLASPARTYL FLUOROMETHYL KETONE; CASPASE INHIBITOR; COPPER; CREATINE; GLUTAMIC ACID; PERIPHERIN; PROTEIN BCL 2; RILUZOLE; SUPEROXIDE DISMUTASE;

AMYOTROPHIC LATERAL SCLEROSIS; APOPTOSIS; AUTOIMMUNITY; CATALYSIS; GENE MUTATION; HUMAN; NEUROFILAMENT; NONHUMAN; PRIORITY JOURNAL; REVIEW;

EID: 0035936804     PISSN: 00928674     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0092-8674(01)00244-6     Document Type: Review

References (75)

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14. Elevated free nitrotyrosine levels, but not protein-bound nitrotyrosine or hydroxyl radicals, throughout amyotrophic lateral sclerosis (ALS)-like disease implicate tyrosine nitration as an aberrant in vivo property of one familial ALS-linked superoxidase dismutase 1 mutant
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