Central role for NMDA receptors in redox mediated impairment of synaptic function during aging and Alzheimer's disease

Behavioural Brain Research

Volumn 322, Issue , 2017, Pages 223-232

  Foster, T C ^a   Kyritsopoulos, C ^a   Kumar, A ^a  

^a UNIVERSITY OF FLORIDA COLLEGE OF MEDICINE   (United States)

Author keywords

Aging; Alzheimer's disease; Calcium dysregulation; Cognitive function; Hippocampus; Intracellular calcium stores; NMDA receptor; Synaptic plasticity

Indexed keywords

AMYLOID BETA PROTEIN; CALCIUM ION; N METHYL DEXTRO ASPARTIC ACID RECEPTOR; PRESENILIN 1;

AGING; ALZHEIMER DISEASE; ARTICLE; BRAIN DYSFUNCTION; CALCIUM TRANSPORT; CELL AGING; COGNITIVE DEFECT; EXECUTIVE FUNCTION; HIGH RISK POPULATION; HUMAN; MEMORY DISORDER; MENTAL PERFORMANCE; MOLECULAR PATHOLOGY; NERVE CELL PLASTICITY; NEUROPATHOLOGY; NONHUMAN; OXIDATION REDUCTION STATE; OXIDATIVE STRESS; PROTEIN DEGRADATION; PROTEIN FUNCTION; RISK ASSESSMENT; SYNAPSE; SYNAPTIC TRANSMISSION; ANIMAL; METABOLISM; OXIDATION REDUCTION REACTION; PHYSIOLOGY;

AGING; ALZHEIMER DISEASE; ANIMALS; HUMANS; OXIDATION-REDUCTION; RECEPTORS, N-METHYL-D-ASPARTATE; SYNAPSES; SYNAPTIC TRANSMISSION;

EID: 84971601466     PISSN: 01664328     EISSN: 18727549     Source Type: Journal    
DOI: 10.1016/j.bbr.2016.05.012     Document Type: Article

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