KRAS mutant pancreatic cancer: No lone path to an effective treatment

Cancers

Volumn 8, Issue 4, 2016, Pages

  Zeitouni, Daniel ^a   Pylayeva Gupta, Yuliya ^a   Der, Channing J ^a   Bryant, Kirsten L ^a  

^a UNIVERSITY OF NORTH CAROLINA   (United States)

Author keywords

Cancer; Pancreatic; RAS; Therapeutics

Indexed keywords

BRYOSTATIN 1; ENZYME INHIBITOR; GLUTAMINE; K RAS PROTEIN; MAMMALIAN TARGET OF RAPAMYCIN; MITOGEN ACTIVATED PROTEIN KINASE; MITOGEN ACTIVATED PROTEIN KINASE KINASE; PHOSPHATIDYLINOSITOL 3 KINASE; PROTEIN FARNESYLTRANSFERASE INHIBITOR; PROTEIN KINASE B; RAF PROTEIN; RAS INHIBITOR; SALIRASIB; SMALL INTERFERING RNA; TIPIFARNIB; UNCLASSIFIED DRUG;

AMINO ACID METABOLISM; AUTOPHAGY; CANCER GROWTH; CANCER MORTALITY; CANCER SURVIVAL; CANCER THERAPY; CELL MEMBRANE; CELLULAR DISTRIBUTION; DRUG EFFICACY; DRUG MECHANISM; DRUG RESEARCH; DRUG TARGETING; ENZYME INHIBITION; ENZYME LOCALIZATION; GENE INTERACTION; GENE MUTATION; GENETIC SCREENING; GLUCOSE UTILIZATION; GLYCOLYSIS; HUMAN; IMMUNE RESPONSE; MUTANT; NONHUMAN; ONCOGENE K RAS; PANCREAS ADENOCARCINOMA; PANCREAS CANCER; PINOCYTOSIS; PROTEIN TARGETING; REVIEW; RNA INTERFERENCE; SIGNAL TRANSDUCTION; SURVIVAL RATE; SURVIVAL TIME;

EID: 84964282698     PISSN: None     EISSN: 20726694     Source Type: Journal    
DOI: 10.3390/cancers8040045     Document Type: Review

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