The MRE11 complex: An important source of stress relief

Experimental Cell Research

Volumn 329, Issue 1, 2014, Pages 162-169

  Rein, Katrin ^a   Stracker, Travis H ^a  

^a INSTITUTE FOR RESEARCH IN BIOMEDICINE   (Spain)

Author keywords

Apoptosis; Cancer; Cell cycle checkpoint; DNA damage; DNA repair; Genomic instability; MRE11; NBS1; RAD50; Replication fork

Indexed keywords

APHIDICOLIN; DNA; MRE11 PROTEIN; NIBRIN; NUCLEASE; RAD50 PROTEIN; TUMOR SUPPRESSOR PROTEIN; ATM PROTEIN; CARRIER PROTEIN; DNA BINDING PROTEIN; MRE11A PROTEIN, HUMAN; NUCLEAR PROTEIN; TOPBP1 PROTEIN, HUMAN;

CANCER INHIBITION; COMPLEX FORMATION; DNA DAMAGE; DNA REPAIR; DNA REPLICATION; DOUBLE STRANDED DNA BREAK; GENE DELETION; HUMAN; NONHUMAN; PROTEIN DOMAIN; PROTEIN FUNCTION; PROTEIN PROTEIN INTERACTION; REVIEW; SIGNAL TRANSDUCTION; CELL TRANSFORMATION; GENETICS; METABOLISM; PATHOLOGY;

ATAXIA TELANGIECTASIA MUTATED PROTEINS; CARRIER PROTEINS; CELL TRANSFORMATION, NEOPLASTIC; DNA REPLICATION; DNA-BINDING PROTEINS; HUMANS; NUCLEAR PROTEINS;

EID: 84919346374     PISSN: 00144827     EISSN: 10902422     Source Type: Journal    
DOI: 10.1016/j.yexcr.2014.10.010     Document Type: Review

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