CtIP maintains stability at common fragile sites and inverted repeats by end resection-independent endonuclease activity

Molecular Cell

Volumn 54, Issue 6, 2014, Pages 1012-1021

  Wang, Hailong ^a   Li, Yongjiang ^a   Truong, Lan N ^a   Shi, Linda Z ^b   Hwang, PattyYi Hwa ^a   He, Jing ^a   Do, Johnny ^a   Cho, Michael Jeffrey ^a   Li, Hongzhi ^d   Negrete, Alejandro ^e   Shiloach, Joseph ^e   Berns, Michael W ^b,c   Shen, Binghui ^d   Chen, Longchuan ^f   Wu, Xiaohua ^a  

^a SCRIPPS RESEARCH INSTITUTE   (United States)

^b UNIVERSITY OF CALIFORNIA   (United States)

^c UNIVERSITY OF CALIFORNIA   (United States)

^d CITY OF HOPE NATIONAL MEDICAL CENTER   (United States)

^e NATIONAL INSTITUTE OF DIABETES AND DIGESTIVE AND KIDNEY DISEASES   (United States)

^f VA LONG BEACH HEALTHCARE SYSTEM   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

BINDING PROTEIN; ENDONUCLEASE; MUTANT PROTEIN; PROTEIN CTIP; PROTEIN MRN; UNCLASSIFIED DRUG;

ALU SEQUENCE; ARTICLE; AT RICH SEQUENCE; CHROMOSOME FRAGILE SITE; CONTROLLED STUDY; DNA STRUCTURE; DOUBLE STRANDED DNA BREAK; ENZYME ACTIVITY; GENE LOCUS; HOMOLOGOUS RECOMBINATION; HUMAN; HUMAN CELL; INVERTED REPEAT; MAMMAL CELL; MITOTIC RECOMBINATION; PROTEIN STABILITY;

MAMMALIA;

ALU ELEMENTS; BASE COMPOSITION; CARRIER PROTEINS; CELL CYCLE PROTEINS; CELL LINE; CHROMOSOME FRAGILE SITES; DNA BREAKS, DOUBLE-STRANDED; DNA REPAIR; DNA REPAIR ENZYMES; DNA-BINDING PROTEINS; ENDONUCLEASES; HOMOLOGOUS RECOMBINATION; HUMANS; INVERTED REPEAT SEQUENCES; MITOSIS; NUCLEAR PROTEINS; RECOMBINATION, GENETIC;

EID: 84903581320     PISSN: 10972765     EISSN: 10974164     Source Type: Journal    
DOI: 10.1016/j.molcel.2014.04.012     Document Type: Article

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